Alison Sinclair
#93,527
Most Influential Person Now
Researcher
Alison Sinclair 's AcademicInfluence.com Rankings
Alison Sinclair computer-science Degrees
Computer Science
#12293
World Rank
#13120
Historical Rank
Machine Learning
#5639
World Rank
#5715
Historical Rank
Artificial Intelligence
#6087
World Rank
#6181
Historical Rank
Database
#9368
World Rank
#9862
Historical Rank
Download Badge
Computer Science
Alison Sinclair 's Degrees
- PhD Computer Science Stanford University
- Masters Computer Science University of California, Berkeley
- Bachelors Computer Science University of California, Berkeley
Similar Degrees You Can Earn
Why Is Alison Sinclair Influential?
(Suggest an Edit or Addition)According to Wikipedia, Alison Jane Sinclair is professor of molecular virology at the University of Sussex where she runs the Sinclair Lab. Her research interests include the Epstein Barr virus. She is a fellow of the Royal Society of Biology and a senior fellow of the Higher Education Academy.
Alison Sinclair 's Published Works
Published Works
- Synthetic peptide homologous to beta protein from Alzheimer disease forms amyloid-like fibrils in vitro. (1987) (461)
- A thyroid‐specific nuclear protein essential for tissue‐specific expression of the thyroglobulin promoter. (1989) (369)
- Thyroid-specific gene expression (1994) (153)
- Pathways of activation of the Epstein-Barr virus productive cycle (1991) (139)
- bZIP proteins of human gammaherpesviruses. (2003) (104)
- Bioimpedance Analysis for the Characterization of Breast Cancer Cells in Suspension (2012) (96)
- The tissue-specific expression of the thyroglobulin gene requires interaction between thyroid-specific and ubiquitous factors. (1990) (94)
- Cyclins D1 and D2 are differentially expressed in human B-lymphoid cell lines. (1993) (85)
- Induction of p16INK4a Is the Major Barrier to Proliferation when Epstein-Barr Virus (EBV) Transforms Primary B Cells into Lymphoblastoid Cell Lines (2013) (68)
- Host cell requirements for efficient infection of quiescent primary B lymphocytes by Epstein-Barr virus (1995) (61)
- Strain variation in Epstein-Barr virus immediate early genes. (1993) (57)
- Epstein-Barr virus exploits the normal cell pathway to regulate Rb activity during the immortalisation of primary B-cells. (1996) (56)
- Dynamic Chromatin Environment of Key Lytic Cycle Regulatory Regions of the Epstein-Barr Virus Genome (2011) (55)
- The normal cell cycle activation program is exploited during the infection of quiescent B lymphocytes by Epstein-Barr virus. (1995) (53)
- DNA-damage response pathways triggered by viral replication (2006) (53)
- Mitochondrial DNA mutations in Alzheimer's disease. (1997) (52)
- Epigenetic Control of Viral Life-Cycle by a DNA-Methylation Dependent Transcription Factor (2011) (52)
- Expression of cyclin D2 in Epstein-Barr virus-positive Burkitt's lymphoma cell lines is related to methylation status of the gene (1995) (51)
- Electrical properties of breast cancer cells from impedance measurement of cell suspensions (2010) (49)
- Functional Interaction between Epstein-Barr Virus Replication Protein Zta and Host DNA Damage Response Protein 53BP1 (2009) (45)
- The efficient and rapid import of a peptide into primary B and T lymphocytes and a lymphoblastoid cell line. (1998) (45)
- Methylated DNA Recognition during the Reversal of Epigenetic Silencing Is Regulated by Cysteine and Serine Residues in the Epstein-Barr Virus Lytic Switch Protein (2008) (41)
- Epstein–Barr virus transcription factor Zta acts through distal regulatory elements to directly control cellular gene expression (2015) (41)
- Genome-Wide Analyses of Zta Binding to the Epstein-Barr Virus Genome Reveals Interactions in both Early and Late Lytic Cycles and an Epigenetic Switch Leading to an Altered Binding Profile (2012) (40)
- Mutation of a Single Amino Acid Residue in the Basic Region of the Epstein-Barr Virus (EBV) Lytic Cycle Switch Protein Zta (BZLF1) Prevents Reactivation of EBV from Latency (2005) (39)
- The Epstein–Barr virus lytic cycle activator Zta interacts with methylated ZRE in the promoter of host target gene egr1 (2009) (38)
- Epstein-Barr virus transcription factors. (1992) (36)
- Divergent Requirements for the MAPKERK Signal Transduction Pathway during Initial Virus Infection of Quiescent Primary B Cells and Disruption of Epstein-Barr Virus Latency by Phorbol Esters (1999) (35)
- Thyroid specific gene expression. (1989) (33)
- The Zipper Region of Epstein-Barr Virus bZIP Transcription Factor Zta Is Necessary but Not Sufficient To Direct DNA Binding (2003) (33)
- Reciprocal antagonism of steroid hormones and BZLF1 in switch between Epstein-Barr virus latent and productive cycle gene expression (1992) (31)
- Biophysical Analysis of Natural Variants of the Multimerization Region of Epstein-Barr Virus Lytic-Switch Protein BZLF1 (2001) (30)
- Unexpected structure of Epstein-Barr virus lytic cycle activator Zta. (2006) (26)
- Biophysical and Mutational Analysis of the Putative bZIP Domain of Epstein-Barr Virus EBNA 3C (2004) (24)
- Atypical bZIP Domain of Viral Transcription Factor Contributes to Stability of Dimer Formation and Transcriptional Function (2007) (23)
- Epigenetic control of Epstein–Barr virus transcription – relevance to viral life cycle? (2013) (20)
- The reversal of epigenetic silencing of the EBV genome is regulated by viral bZIP protein. (2008) (19)
- Cell cycle-dependent caspase-like activity that cleaves p27(KIP1) is the beta(1) subunit of the 20S proteasome. (2007) (18)
- Cell cycle arrest following exposure of EBV‐immortalised B‐cells to gamma irradiation correlates with inhibition of cdk2 activity (1998) (15)
- Identification of Epstein-Barr Virus Replication Proteins in Burkitt’s Lymphoma Cells (2015) (15)
- Using Blogs to Enhance Student Engagement and Learning in the Health Sciences. (2013) (14)
- Interactions between Epstein-Barr virus and the cell cycle control machinery. (1998) (14)
- Reduced signal transduction through glucocorticoid receptor in Burkitt's lymphoma cell lines. (1994) (13)
- Repression of CIITA by the Epstein-Barr virus transcription factor Zta is independent of its dimerization and DNA binding. (2016) (12)
- [6] Methods to study epstein-barr virus and p53 status in human cells (1995) (12)
- Is EBV Associated with Breast Cancer in Specific Geographic Locations? (2021) (12)
- Evaluation of a Prediction Protocol to Identify Potential Targets of Epigenetic Reprogramming by the Cancer Associated Epstein Barr Virus (2010) (12)
- p16(INK4A)-independence of Epstein-Barr virus-induced cell proliferation and virus latency. (2004) (9)
- The deoxyribonucleic acid regions involved in the hormonal regulation of thyroglobulin gene expression. (1991) (9)
- Implementing Service Learning: From Nutrition Education into Community Action (2011) (8)
- Investigation of the Multimerization Region of the Kaposi's Sarcoma-Associated Herpesvirus (Human Herpesvirus 8) Protein K-bZIP: the Proposed Leucine Zipper Region Encodes a Multimerization Domain with an Unusual Structure (2005) (7)
- Identifying the Cellular Interactome of Epstein-Barr Virus Lytic Regulator Zta Reveals Cellular Targets Contributing to Viral Replication (2019) (7)
- Correction: Induction of p16INK4a Is the Major Barrier to Proliferation when Epstein-Barr Virus (EBV) Transforms Primary B Cells into Lymphoblastoid Cell Lines (2013) (7)
- KIPase activity is a novel caspase-like activity associated with cell proliferation. (2004) (6)
- The Epstein – Barr virus lytic cycle activator Zta interacts with methylated ZRE in the promoter of host target gene egr 1 (2009) (4)
- [Effects of Epstein-Barr virus on host gene expression in Burkitt's lymphoma cell lines]. (2009) (4)
- Mechanism of activation of the BNLF2a immune evasion gene of Epstein-Barr virus by Zta (2018) (4)
- Using Service Learning As A Method Of Transferring Health Knowledge (2013) (3)
- Starting An ePortfolio: A Multi-Disciplinary Approach (2014) (3)
- The Use of Chromatin Precipitation Coupled to DNA Sequencing (ChIP-Seq) for the Analysis of Zta Binding to the Human and EBV Genome. (2017) (2)
- Functional Interaction between Epstein-Barr Virus Replication Protein Zta and Host DNA Damage Response Protein 53 BP 1 † (2009) (1)
- Service learning in nutrition education (2008) (1)
- Could Changing the DNA Methylation Landscape Promote the Destruction of Epstein-Barr Virus-Associated Cancers? (2021) (1)
- Gene Expression in Burkitt’s Lymphoma Cells (1994) (1)
- Communication p 16 INK 4 A-independence of Epstein – Barr virus-induced cell proliferation and virus latency (2004) (1)
- Interaction sites of the Epstein–Barr virus Zta transcription factor with the host genome in epithelial cells (2021) (1)
- Functional Interaction between Epstein-Barr Virus Replication Protein Zta and Host DNA Damage Response Protein 53BP1 (cid:1) (2009) (0)
- Review bZIP proteins of human gammaherpesviruses (2003) (0)
- Research Capsule Epstein-Barr Vi rus Transcription Factors (2005) (0)
- Comparison of amyloid from Alzheimer’s disease with synthetic peptide (1988) (0)
- Cell cycle distribution of B-lymphocytes and cell lines. (2001) (0)
- The import of peptides into lymphocytes and their effects on the cell cycle (1997) (0)
- Poster Session - Implementing Service Learning: From Nutrition Education into Student Action (2011) (0)
- What about our talented students: an exploratory study (2010) (0)
- Using Service Learning to Create Advocacy for Nutrition Education (2008) (0)
- Enhancing Students’ Confidence in Employability Skills through the Practice of “Recall, Adapt and Apply” (2017) (0)
- Methylated DNA recognition during the reversal of epigenetic silencing is regulated by cysteine and cerine residues in the (2019) (0)
- Down regulation of the cyclin-dependent kinase inhibitor p27Kip1 by Epstein-Barr virus (1997) (0)
- Review bZIP proteins of human gammaherpesviruses (2003) (0)
- University of Dundee Mechanism of activation of the BNLF 2 a immune evasion gene of Epstein-Barr virus (2018) (0)
- Review bZIP proteins of human gammaherpesviruses (2003) (0)
- Epstein-Barr Vi rus Transcription Factors (1992) (0)
- Virus the Infection of Quiescent B Lymphocytes by Epstein-Barr The Normal Cell Cycle Activation Program Is Exploited during Updated (2006) (0)
- Mechanism of activation of the BNLF 2 a immune evasion gene of (2019) (0)
This paper list is powered by the following services:
Other Resources About Alison Sinclair
What Schools Are Affiliated With Alison Sinclair ?
Alison Sinclair is affiliated with the following schools: