Christopher J. Marshall

Christopher J. Marshall's AcademicInfluence.com Rankings

Biology

#15998

World Rank

#20098

Historical Rank

Cell Biology

#901

World Rank

#925

Historical Rank

Biochemistry

#2811

World Rank

#3007

Historical Rank

Neuroscience

#2929

World Rank

#3008

Historical Rank

biology Degrees

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Biology

Christopher J. Marshall's Degrees

PhD Biochemistry University of California, San Francisco
Bachelors Chemistry Stanford University

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Christopher J. Marshall's Published Works

Number of citations in a given year to any of this author's works

Total number of citations to an author for the works they published in a given year. This highlights publication of the most important work(s) by the author

Published Works

Sustained activation of the mitogen-activated protein (MAP) kinase cascade may be required for differentiation of PC12 cells. Comparison of the effects of nerve growth factor and epidermal growth factor. (1992) (890)
Stimulation of the stress-activated mitogen-activated protein kinase subfamilies in perfused heart. p38/RK mitogen-activated protein kinases and c-Jun N-terminal kinases are activated by ischemia/reperfusion. (1996) (561)
Identification of the sites in MAP kinase kinase‐1 phosphorylated by p74raf‐1. (1994) (539)
A CAAX or a CAAL motif and a second signal are sufficient for plasma membrane targeting of ras proteins. (1991) (451)
MAPKAP kinase‐2; a novel protein kinase activated by mitogen‐activated protein kinase. (1992) (450)
Endothelin-1 and fibroblast growth factors stimulate the mitogen-activated protein kinase signaling cascade in cardiac myocytes. The potential role of the cascade in the integration of two signaling pathways leading to myocyte hypertrophy. (1994) (385)
Activation of extracellular signal‐regulated kinase, ERK2, by p21ras oncoprotein. (1992) (378)
Post‐translational processing of p21ras is two‐step and involves carboxyl‐methylation and carboxy‐terminal proteolysis. (1989) (373)
Dynamic fatty acylation of p21N‐ras. (1987) (262)
A single autophosphorylation site confers oncogenicity to the Neu/ErbB‐2 receptor and enables coupling to the MAP kinase pathway. (1994) (213)
The mitogen-activated protein (MAP) kinase cascade can either stimulate or inhibit DNA synthesis in primary cultures of rat hepatocytes depending upon whether its activation is acute/phasic or chronic. (1998) (196)
Identification of novel phosphorylation sites required for activation of MAPKAP kinase‐2. (1995) (189)
Assay and expression of mitogen-activated protein kinase, MAP kinase kinase, and Raf. (1995) (169)
Proliferative but not nonproliferative responses to granulocyte colony-stimulating factor are associated with rapid activation of the p21ras/MAP kinase signalling pathway. (1994) (118)
Beta interferon and oncostatin M activate Raf-1 and mitogen-activated protein kinase through a JAK1-dependent pathway (1997) (116)
Specific association of activated MAP kinase kinase kinase (Raf) with the plasma membranes of ras-transformed retinal cells. (1993) (95)
Activation of the Raf/MAP kinase cascade by the Ras‐related protein TC21 is required for the TC21‐mediated transformation of NIH 3T3 cells (1999) (81)
Interleukin 1 and tumour necrosis factor activate the mitogen‐activated protein (MAP) kinase kinase in cultured cells (1993) (71)
A scintillation proximity assay for the Raf/MEK/ERK kinase cascade: high-throughput screening and identification of selective enzyme inhibitors. (1999) (67)
The amino acid sequence of a mammalian MAP kinase kinase. (1992) (60)
p21H‐ras‐induced morphological transformation and increases in c‐myc expression are independent of functional protein kinase C. (1989) (57)
The role of MAP kinase kinase in interleukin-3 stimulation of proliferation. (1996) (53)
The threonine residues in MAP kinase kinase 1 phosphorylated by MAP kinase in vitro are also phosphorylated in nerve growth factor‐stimulated rat phaeochromocytoma (PC12) cells (1994) (35)
An in vitro 96-well plate assay of the mitogen-activated protein kinase cascade. (1999) (20)
Mitogen-activated protein (MAP) kinase stimulation by phorbol esters and external load in the isolated perfused heart. (1993) (4)
Acidic fibroblast growth factor or endothelin-1 stimulate the MAP kinase cascade in cardiac myocytes. (1993) (3)
Enhanced ERα, ERBB2 and MAPK signal transduction pathways operate during the adaptation of MCF-7 cells to long term oestrogen deprivation (2003) (1)

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What Schools Are Affiliated With Christopher J. Marshall?

Christopher J. Marshall is affiliated with the following schools:

Institute of Cancer Research

Christopher J. Marshall's Academic­Influence.com Rankings

Christopher J. Marshall's Degrees

Why Is Christopher J. Marshall Influential?

Christopher J. Marshall's Published Works

Published Works

What Schools Are Affiliated With Christopher J. Marshall?

Christopher J. Marshall's AcademicInfluence.com Rankings