Diane Lipscombe
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Physiologist
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Diane Lipscombebiology Degrees
Biology
#5485
World Rank
#7977
Historical Rank
#1891
USA Rank
Physiology
#517
World Rank
#769
Historical Rank
#68
USA Rank
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Biology
Diane Lipscombe's Degrees
- PhD Physiology University of Oxford
- Bachelors Physiology University of Liverpool
Why Is Diane Lipscombe Influential?
(Suggest an Edit or Addition)According to Wikipedia, Diane Lipscombe is a British neuroscientist who is a professor of neuroscience and the Reliance Dhirubhai Ambani Director of Brown University’s Robert J. and Nancy D. Carney Institute for Brain Science. She served as the president of the Society for Neuroscience in 2019, the world’s largest organization for the study of the brain and nervous system.
Diane Lipscombe's Published Works
Published Works
- Multiple types of neuronal calcium channels and their selective modulation (1988) (1428)
- Neuronal CaV1.3α1 L-Type Channels Activate at Relatively Hyperpolarized Membrane Potentials and Are Incompletely Inhibited by Dihydropyridines (2001) (558)
- α-Adrenergic inhibition of sympathetic neurotransmitter release mediated by modulation of N-type calcium-channel gating (1989) (439)
- L-type calcium channels: the low down. (2004) (430)
- Imaging of cytosolic Ca2+ transients arising from Ca2+ stores and Ca2+ channels in sympathetic neurons (1988) (304)
- Spatial distribution of calcium channels and cytosolic calcium transients in growth cones and cell bodies of sympathetic neurons. (1988) (289)
- Cell-Specific Alternative Splicing Increases Calcium Channel Current Density in the Pain Pathway (2004) (226)
- Identification of Functionally Distinct Isoforms of the N-Type Ca2+ Channel in Rat Sympathetic Ganglia and Brain (1997) (199)
- Neuronal L-Type Calcium Channels Open Quickly and Are Inhibited Slowly (2005) (190)
- Alternative splicing controls G protein–dependent inhibition of N-type calcium channels in nociceptors (2007) (167)
- Reflections on Ca2+-channel diversity, 1988–1994 (1995) (161)
- Neuronal proteins custom designed by alternative splicing (2005) (158)
- Differential Role of N-Type Calcium Channel Splice Isoforms in Pain (2007) (149)
- Alternative splicing: functional diversity among voltage-gated calcium channels and behavioral consequences. (2013) (102)
- Functional diversity in neuronal voltage-gated calcium channels by alternative splicing of Cavα1 (2002) (95)
- Multiple modes of N-type calcium channel activity distinguished by differences in gating kinetics (1993) (90)
- Opioid inhibition of N-type Ca2+ channels and spinal analgesia couple to alternative splicing (2010) (89)
- Control of neuronal voltage-gated calcium ion channels from RNA to protein (2013) (82)
- Alternative splicing in the C‐terminus of CaV2.2 controls expression and gating of N‐type calcium channels (2006) (79)
- L-type calcium channels: highs and new lows. (2002) (75)
- Retrograde Synaptic Inhibition Is Mediated by α-Neurexin Binding to the α2δ Subunits of N-Type Calcium Channels (2017) (73)
- The inhibition of neuronal calcium ion channels by trace levels of yttrium released from carbon nanotubes. (2009) (72)
- Alternative splicing in the voltage-sensing region of N-Type CaV2.2 channels modulates channel kinetics. (2004) (70)
- Neuronal calcium channels: splicing for optimal performance. (2007) (69)
- Alternative Splicing in the Cytoplasmic II–III Loop of the N-Type Ca Channel α1B Subunit: Functional Differences Are β Subunit-Specific (2000) (66)
- CACNA1B mutation is linked to unique myoclonus-dystonia syndrome. (2015) (66)
- Cumulative inactivation of N-type CaV2.2 calcium channels modified by alternative splicing. (2004) (50)
- A rare schizophrenia risk variant of CACNA1I disrupts CaV3.3 channel activity (2016) (49)
- The neuronal splicing factor Nova controls alternative splicing in N-type and P-type CaV2 calcium channels (2010) (44)
- The N‐Type Ca Channel in Frog Sympathetic Neurons and Its Role in α‐Adrenergic Modulation of Transmitter Release (1989) (42)
- Alternative splicing in the cytoplasmic II-III loop of the N-type Ca channel alpha 1B subunit: functional differences are beta subunit-specific. (2000) (40)
- Differential Ubiquitination and Proteasome Regulation of CaV2.2 N-Type Channel Splice Isoforms (2012) (39)
- Nicotinic receptors of frog ganglia resemble pharmacologically those of skeletal muscle (1988) (37)
- Calcium Channel CaVα₁ Splice Isoforms - Tissue Specificity and Drug Action. (2015) (34)
- Spinal morphine but not ziconotide or gabapentin analgesia is affected by alternative splicing of voltage-gated calcium channel CaV2.2 pre-mRNA (2013) (34)
- Calcium channel activation stabilizes a neuronal calcium channel mRNA (1999) (34)
- Novel CaV2.1 clone replicates many properties of Purkinje cell CaV2.1 current (2007) (31)
- The alpha(2)delta subunit augments functional expression and modifies the pharmacology of Ca(V)1.3 L-type channels. (2009) (30)
- Decreased microRNA levels lead to deleterious increases in neuronal M2 muscarinic receptors in Spinal Muscular Atrophy models (2017) (29)
- Circuit Dysfunction in SOD1-ALS Model First Detected in Sensory Feedback Prior to Motor Neuron Degeneration Is Alleviated by BMP Signaling (2018) (29)
- Alternative Splicing Matters: N-Type Calcium Channels in Nociceptors (2007) (26)
- Alternative splicing of neuronal genes: new mechanisms and new therapies (2019) (25)
- Single‐channel analysis of a point mutation of a conserved serine residue in the S2 ligand‐binding domain of the NR2A NMDA receptor subunit (2006) (25)
- Repressing the neuron within. (2008) (22)
- The molecular identity of Ca channel alpha 1-subunits expressed in rat sympathetic neurons. (1996) (22)
- Constitutive activity of the Ghrelin receptor reduces surface expression of voltage-gated Ca2+ channels in a CaVβ-dependent manner (2017) (22)
- Alternative Splicing in Voltage Gated Calcium Channels (2004) (20)
- Cell-specific exon methylation and CTCF binding in neurons regulate calcium ion channel splicing and function (2020) (19)
- Cacna1b alternative splicing impacts excitatory neurotransmission and is linked to behavioral responses to aversive stimuli (2019) (16)
- The molecular identity of Ca channel α1-subunits expressed in rat sympathetic neurons (2007) (16)
- Cell-Specific RNA Binding Protein Rbfox2 Regulates CaV2.2 mRNA Exon Composition and CaV2.2 Current Size (2017) (14)
- Meta-analysis of Genetic Modifiers Reveals Candidate Dysregulated Pathways in Amyotrophic Lateral Sclerosis (2019) (14)
- Framework for advancing rigorous research (2020) (12)
- The α 2 δ subunit augments functional expression and modifies the pharmacology of CaV1.3 L-type channels (2010) (9)
- Internalizing channels: a mechanism to control pain? (2006) (8)
- Selective control of synaptically-connected circuit elements by all-optical synapses (2021) (8)
- Long-Term Depression Induced by Optogenetically Driven Nociceptive Inputs to Trigeminal Nucleus Caudalis or Headache Triggers (2018) (7)
- Epigenetic control of ion channel expression and cell-specific splicing in nociceptors: Chronic pain mechanisms and potential therapeutic targets (2020) (6)
- Exposure to space radiation of high-performance infrared multilayer filters and materials technology experiments (A0056) (1984) (5)
- Cell-specific exon methylation and CTCF binding in neurons regulates calcium ion channel splicing and function (2019) (5)
- Alternative Splicing of Neuronal Cav2 Calcium Channels (2008) (5)
- Analysing an allelic series of rare missense variants of CACNA1I in a Swedish schizophrenia cohort (2021) (4)
- Tripping the HCN Breaker (2009) (4)
- Heat But Not Mechanical Hypersensitivity Depends on Voltage-Gated CaV2.2 Calcium Channel Activity in Peripheral Axon Terminals Innervating Skin (2021) (4)
- Biophysics of Voltage-Gated Ion Channels (2014) (4)
- Miniaturized Devices for Bioluminescence Imaging in Freely Behaving Animals (2020) (3)
- Author response: Decreased microRNA levels lead to deleterious increases in neuronal M2 muscarinic receptors in Spinal Muscular Atrophy models (2017) (2)
- Bioluminescent optogenetic (BL-OG) activation of neurons during mouse postnatal brain development (2021) (2)
- Bioluminescence-Driven Optogenetics (2017) (2)
- Editorial Overview: Ion Channels (2018) (2018) (2)
- Tracks through the genome to physiological events (2015) (2)
- Peripheral voltage-gated calcium channels in skin are essential for transient neurogenic thermal hyperalgesia in mice (2020) (1)
- 2.2 Channels Modulates Channel Kinetics V N-Type Ca Alternative Splicing in the Voltage-Sensing Region of (2015) (0)
- Protected by a Fox (2018) (0)
- Decision letter: Novel charged sodium and calcium channel inhibitor active against neurogenic inflammation (2019) (0)
- There and back again: G proteins inhibit neuronal calcium ion channels (2012) (0)
- Alternative splicing and calcium ion channel diversity (2011) (0)
- Constitutive activity of the Ghrelin receptor reduces surface expression of voltage-gated Ca<sup>2+</sup> channels in a Ca<sub>V</sub>β-dependent manner (2017) (0)
- Cacna1b alternative splicing impacts excitatory neurotransmission and is linked to behavioral responses to aversive stimuli (2019) (0)
- Ion Channels (2021) (0)
- DifferentialUbiquitinationandProteasomeRegulationof Ca V 2.2N-TypeChannelSpliceIsoforms (2012) (0)
- North West Shelf LNG project (1979) (0)
- S17 Ion channels, neuromodulators and ganglionic regulation: S17.1 Nociceptors regulate calcium channel drug sensitivity using alternative splicing (2009) (0)
- α-Adrenergic inhibition of N-type calcium channels and neurotransmitter release in sympathetic neurones (1990) (0)
- Author response: Cell-specific exon methylation and CTCF binding in neurons regulate calcium ion channel splicing and function (2020) (0)
- Developing biolight-based molecular technologies for simultaneous mapping, imaging, and controlling neural activity (2021) (0)
- SWNTs inhibit normal physiological function of calcium ion channels through yttrium release (2008) (0)
- Author Proof A Repressing the neuron within (2007) (0)
- Isoform of human calcium channel type N and uses thereof (1999) (0)
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What Schools Are Affiliated With Diane Lipscombe?
Diane Lipscombe is affiliated with the following schools: