Diane Reay
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Sociology
Why Is Diane Reay Influential?
(Suggest an Edit or Addition)According to Wikipedia, Diane Reay is a sociologist and academic, who is Professor of Education at the University of Cambridge. She is noted for her study about educational inequalities among students in state schools in the United Kingdom. She has maintained that there is a tendency to misuse the school selection practice to transform social class differences into education. For instance, she criticized the Oxbridge application process as "institutionally racist".
Diane Reay's Published Works
Published Works
- NF-κB inhibition delays DNA damage-induced senescence and aging in mice. (2012) (353)
- Inhibition of the IKK/NF-κB pathway by AAV gene transfer improves muscle regeneration in older mdx mice (2010) (52)
- Role of UCHL1 in axonal injury and functional recovery after cerebral ischemia (2019) (43)
- Systemic delivery of NEMO binding domain/IKKγ inhibitory peptide to young mdx mice improves dystrophic skeletal muscle histopathology (2011) (43)
- Local high-capacity adenovirus-mediated mCTLA4Ig and mCD40Ig expression prolongs recombinant gene expression in skeletal muscle. (2001) (40)
- Full-length dystrophin gene transfer to the mdx mouse in utero (2008) (38)
- Improvement of the mdx mouse dystrophic phenotype by systemic in utero AAV8 delivery of a minidystrophin gene (2010) (35)
- Comparison of high-capacity and first-generation adenoviral vector gene delivery to murine muscle in utero (2005) (28)
- Fetal muscle gene transfer is not enhanced by an RGD capsid modification to high-capacity adenoviral vectors (2003) (27)
- Development of novel NEMO-binding domain mimetics for inhibiting IKK/NF-κB activation (2018) (23)
- Patterns of gene expression from in utero delivery of adenoviral-associated vector serotype 1. (2005) (20)
- Binding of adenoviral fiber knob to the coxsackievirus-adenovirus receptor is crucial for transduction of fetal muscle. (2003) (13)
- Abolishing UCHL1's hydrolase activity exacerbates TBI-induced axonal injury and neuronal death in mice (2020) (7)
- d-Amino Acid Substitution of Peptide-Mediated NF-κB Suppression in mdx Mice Preserves Therapeutic Benefit in Skeletal Muscle, but Causes Kidney Toxicity (2015) (6)
- Effect of Nuclear Factor κB Inhibition on Serotype 9 Adeno-Associated Viral (AAV9) Minidystrophin Gene Transfer to the mdx Mouse (2012) (5)
- Biocompatibility of adenoviral vectors in poly(vinyl chloride) tubing catheters with presence or absence of plasticizer di-2-ethylhexyl phthalate. (2004) (4)
- Improving the identification of patients with a genetic diagnosis of familial hypercholesterolaemia in primary care: A strategy to achieve the NHS long term plan. (2021) (3)
- Gene therapy for muscular dystrophy reaches human clinical trial (2009) (2)
- Mutation of a Ubiquitin Carboxy Terminal Hydrolase L1 Lipid Binding Site Alleviates Cell Death, Axonal Injury, and Behavioral Deficits After Traumatic Brain Injury in Mice (2021) (1)
- G.P.364 D-amino acid substitution of peptide-mediated NF-κB suppression in mdx mice preserves therapeutic benefit in skeletal muscle, but causes kidney toxicity (2015) (0)
- 915. High and Low Affinity Consensus Binding Sequences to Extracellular α7 Integrin X2B Subunit Detected Using Phage Display Biopanning (2006) (0)
- 382. In Utero Intramuscular Delivery of a High-Capacity Adenoviral Vector Carrying a Full-Length Murine Dystrophin cDNA Provides Functional Benefit and Reassembly of the Dystrophin-Glycoprotein Complex in Hind Limb Muscles of mdx Mice (2004) (0)
- 1086. Patterns of Gene Expression from In Utero Delivery of AAV Serotype 1 (2005) (0)
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