Patrick Fitzgerald
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Most Influential Person Now
Patrick Fitzgerald's AcademicInfluence.com Rankings
Patrick Fitzgeraldbiology Degrees
Biology
#16007
World Rank
#20108
Historical Rank
Cell Biology
#902
World Rank
#926
Historical Rank
Genetics
#1912
World Rank
#2032
Historical Rank
Molecular Biology
#2815
World Rank
#2860
Historical Rank

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Biology
Patrick Fitzgerald's Degrees
- Bachelors Biochemistry University of California, Berkeley
Why Is Patrick Fitzgerald Influential?
(Suggest an Edit or Addition)Patrick Fitzgerald's Published Works
Number of citations in a given year to any of this author's works
Total number of citations to an author for the works they published in a given year. This highlights publication of the most important work(s) by the author
Published Works
- The transcription factor Myc controls metabolic reprogramming upon T lymphocyte activation. (2011) (1581)
- Catalytic activity of the caspase-8-FLIPL complex inhibits RIPK3-dependent necrosis (2011) (1069)
- Microtubule-associated protein 1 light chain 3 alpha (LC3)-associated phagocytosis is required for the efficient clearance of dead cells (2011) (573)
- Disruption of Mitochondrial Function during Apoptosis Is Mediated by Caspase Cleavage of the p75 Subunit of Complex I of the Electron Transport Chain (2004) (569)
- p53 Induces Apoptosis by Caspase Activation through Mitochondrial Cytochrome c Release* (2000) (546)
- ESCRT-III Acts Downstream of MLKL to Regulate Necroptotic Cell Death and Its Consequences (2017) (403)
- Enhanced bacterial clearance and sepsis resistance in caspase-12-deficient mice (2006) (333)
- Cell death and tissue remodeling in planarian regeneration. (2010) (300)
- The cytotoxic cell protease granzyme B initiates apoptosis in a cell‐free system by proteolytic processing and activation of the ICE/CED‐3 family protease, CPP32, via a novel two‐step mechanism. (1996) (294)
- GAPDH and Autophagy Preserve Survival after Apoptotic Cytochrome c Release in the Absence of Caspase Activation (2007) (284)
- Characterization of RIPK3-mediated phosphorylation of the activation loop of MLKL during necroptosis (2015) (254)
- Different mitochondrial intermembrane space proteins are released during apoptosis in a manner that is coordinately initiated but can vary in duration. (2006) (235)
- GAPDH and Autophagy Preserve Survival after Apoptotic Cytochrome c Release in the Absence of Caspase Activation (2007) (212)
- Caspase-8 mediates caspase-1 processing and innate immune defense in response to bacterial blockade of NF-κB and MAPK signaling (2014) (205)
- BID-induced structural changes in BAK promote apoptosis (2013) (182)
- Cutting Edge: Endoplasmic Reticulum Stress Licenses Macrophages To Produce Mature IL-1β in Response to TLR4 Stimulation through a Caspase-8– and TRIF-Dependent Pathway (2014) (146)
- Defective Cytochrome c-dependent Caspase Activation in Ovarian Cancer Cell Lines due to Diminished or Absent Apoptotic Protease Activating Factor-1 Activity* (2001) (139)
- Activation-induced T-cell death is cell cycle dependent and regulated by cyclin B (1995) (130)
- Non-apoptotic role of BID in inflammation and innate immunity (2011) (127)
- Role of proteolysis in caspase-8 activation and stabilization. (2007) (123)
- Mitochondrial pathway of apoptosis is ancestral in metazoans (2012) (106)
- Growth inhibition by CDK-cyclin and PCNA binding domains of p21 occurs by distinct mechanisms and is regulated by ubiquitin-proteasome pathway (1999) (103)
- p21 contains independent binding sites for cyclin and cdk2: both sites are required to inhibit cdk2 kinase activity. (1996) (102)
- A conserved domain of the large subunit of replication factor C binds PCNA and acts like a dominant negative inhibitor of DNA replication in mammalian cells. (1996) (94)
- Growth inhibition by CDK-cyclin and PCNA binding domains of p21 occurs by distinct mechanisms and is regulated by ubiquitin-proteasome pathway (1999) (61)
- Role for Cyclin A-dependent Kinase in DNA Replication in Human S Phase Cell Extracts* (1996) (57)
- Caspase-8-Dependent Inflammatory Responses Are Controlled by Its Adaptor, FADD, and Necroptosis. (2020) (55)
- Just So Stories about the Evolution of Apoptosis (2016) (52)
- Noncanonical function of an autophagy protein prevents spontaneous Alzheimer’s disease (2020) (43)
- Inhibition of Hsp90 via 17-DMAG induces apoptosis in a p53-dependent manner to prevent medulloblastoma (2009) (35)
- Nab2 regulates secondary CD8+ T-cell responses through control of TRAIL expression. (2012) (24)
- Presence of a transcriptionally active glucocorticoid receptor α in Lens epithelial cells (2003) (20)
- Sublethal cytochrome c release generates drug-tolerant persister cells (2022) (16)
- Presence of a transcriptionally active glucocorticoid receptor alpha in lens epithelial cells. (2003) (12)
- Yeretssian et al. reply (2012) (3)
- Regulation of an inhibitor of the cell cycle, p21WAF1 CIP1 by the ubiquitin‐proteasome pathway (1999) (1)
- Caspase-8 and FADD prevent spontaneous ZBP1 expression and necroptosis (2022) (1)
- Corrigendum: Enhanced bacterial clearance and sepsis resistance in caspase-12-deficient mice (2013) (1)
- Essential pro-survival role for caspase-8 during T-cell activation and embryonic development through inhibition of RIPK3-dependent necrosis (159.9) (2011) (0)
- Corrigendum: Yeretssian et al. reply (2012) (0)
- TRAIL expression T-cell responses through control of + Nab2 regulates secondary CD8 (2012) (0)
- Lens Epithelial Cells Contain a Transcriptionally Active Alpha Glucocorticoid Receptor (2003) (0)
- Caspase-8, RIPK1 and FADD regulate cell death and caspase-1 activation during yersinia infection (INM3P.438) (2014) (0)
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