Tom Curran

Q: What Schools Are Affiliated With Tom Curran

Tom Curran is affiliated with the following schools: Duke University, University of Pennsylvania, Brown University, Xiamen University, Imperial College London, Ohio State University, University of Oxford, University College London, Icahn School of Medicine at Mount Sinai, University of Kansas, University of Chicago, University of Edinburgh

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Philosophy
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Tom Curran 's Degrees

PhD Pathology University of Oxford

Why Is Tom Curran Influential?

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According to Wikipedia, Thomas Curran FRS is a Scottish medical researcher. He is the Executive Director and Chief Scientific Officer of the Children’s Mercy Research Institute at Children’s Mercy Hospital in Kansas City, Missouri, where he is also the Donald J. Hall Eminent Scholar in Pediatric Research. He is also a Professor of Pediatrics at the University of Missouri-Kansas City School of Medicine and a Professor of Cancer Biology at the University of Kansas School of Medicine. Before taking his current positions in 2016, he was a Professor of Pathology and Laboratory Medicine at the University of Pennsylvania School of Medicine, where he also served as Associate Director of Translational Genomics at the Penn Genome Frontiers Institute.

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Tom Curran 's Published Works

Number of citations in a given year to any of this author's works

Total number of citations to an author for the works they published in a given year. This highlights publication of the most important work(s) by the author

Published Works

Stimulus-transcription coupling in the nervous system: involvement of the inducible proto-oncogenes fos and jun. (1991) (2662)
Prediction of central nervous system embryonal tumour outcome based on gene expression (2002) (2421)
Expression of c-fos protein in brain: metabolic mapping at the cellular level. (1988) (1953)
Mapping patterns of c-fos expression in the central nervous system after seizure. (1987) (1735)
A protein related to extracellular matrix proteins deleted in the mouse mutant reeler (1995) (1691)
Redox regulation of fos and jun DNA-binding activity in vitro. (1990) (1552)
Fos and jun: The AP-1 connection (1988) (1453)
Cross-family dimerization of transcription factors Fos/Jun and ATF/CREB alters DNA binding specificity. (1991) (1252)
Induction of c-fos gene and protein by growth factors precedes activation of c-myc (1984) (1228)
A zinc finger-encoding gene coregulated with c-fos during growth and differentiation, and after cellular depolarization (1988) (1211)
Role of ion flux in the control of c-fos expression (1986) (977)
Stimulus-transcription coupling in neurons: role of cellular immediate-early genes (1989) (964)
Redox activation of Fos‐Jun DNA binding activity is mediated by a DNA repair enzyme. (1992) (887)
Reelin Is a Ligand for Lipoprotein Receptors (1999) (802)
Radial glia cells are candidate stem cells of ependymoma. (2005) (773)
The T-cell transcription factor NFATp is a substrate for calcineurin and interacts with Fos and Jun (1993) (769)
Continuous c-fos expression precedes programmed cell death in vivo (1993) (764)
The Genetic Landscape of the Childhood Cancer Medulloblastoma (2011) (711)
Role of the reelin signaling pathway in central nervous system development. (2001) (671)
Identification and characterization of Ref‐1, a nuclear protein that facilitates AP‐1 DNA‐binding activity. (1992) (667)
Genomics identifies medulloblastoma subgroups that are enriched for specific genetic alterations. (2006) (660)
Scrambler and yotari disrupt the disabled gene and produce a reeler -like phenotype in mice (1997) (647)
Isolation and characterization of the c-fos(rat) cDNA and analysis of post-translational modification in vitro. (1987) (608)
FBJ murine osteosarcoma virus: identification and molecular cloning of biologically active proviral DNA (1982) (566)
Binding of the Wilms' tumor locus zinc finger protein to the EGR-1 consensus sequence. (1990) (550)
Regional and Cellular Patterns of reelin mRNA Expression in the Forebrain of the Developing and Adult Mouse (1998) (549)
Fos-associated protein p39 is the product of the jun proto-oncogene. (1988) (542)
Common DNA binding site for Fos protein complexesand transcription factor AP-1 (1988) (534)
c-fos protein can induce cellular transformation: A novel mechanism of activation of a cellular oncogene (1984) (530)
Regulation of proenkephalin by Fos and Jun. (1989) (527)
Suppression of the Shh pathway using a small molecule inhibitor eliminates medulloblastoma in Ptc1(+/-)p53(-/-) mice. (2004) (520)
The redox/DNA repair protein, Ref-1, is essential for early embryonic development in mice. (1996) (508)
Parallel association of Fos and Jun leucine zippers juxtaposes DNA binding domains. (1989) (504)
Identification of redox/repair protein Ref-1 as a potent activator of p53. (1997) (494)
Targeted disruption of NMDA receptor 1 gene abolishes NMDA response and results in neonatal death (1994) (492)
Reelin Is a Secreted Glycoprotein Recognized by the CR-50 Monoclonal Antibody (1997) (488)
The Fos complex and Fos-related antigens recognize sequence elements that contain AP-1 binding sites. (1988) (478)
fra-1: a serum-inducible, cellular immediate-early gene that encodes a fos-related antigen (1988) (463)
Fos and Jun bind cooperatively to the AP-1 site: reconstitution in vitro. (1988) (449)
Analysis of FBJ-MuSV provirus and c-fos (mouse) gene reveals that viral and cellular fos gene products have different carboxy termini (1983) (446)
Superinduction of c-fos by nerve growth factor in the presence of peripherally active benzodiazepines. (1985) (439)
Recessive resistance to thyroid hormone in mice lacking thyroid hormone receptor beta: evidence for tissue‐specific modulation of receptor function. (1996) (428)
Pro-Leu-Ser/Thr-Pro is a consensus primary sequence for substrate protein phosphorylation. Characterization of the phosphorylation of c-myc and c-jun proteins by an epidermal growth factor receptor threonine 669 protein kinase. (1991) (408)
Isolation of the cyclosporin-sensitive T cell transcription factor NFATp. (1993) (401)
An Enhanced Immune Response in Mice Lacking the Transcription Factor NFAT1 (1996) (394)
Complete nucleotide sequence of a human c-onc gene: deduced amino acid sequence of the human c-fos protein. (1983) (386)
The Hedgehog's tale: developing strategies for targeting cancer (2011) (381)
Disabled-1 acts downstream of Reelin in a signaling pathway that controls laminar organization in the mammalian brain. (1998) (371)
Fos: an immediate-early transcription factor in neurons. (1995) (364)
The redox and DNA-repair activities of Ref-1 are encoded by nonoverlapping domains. (1994) (352)
Loss of p53 but not ARF accelerates medulloblastoma in mice heterozygous for patched. (2001) (344)
Glutamate receptor agonists increase the expression of Fos, Fra, and AP‐1 DNA binding activity in the mammalian brain (1989) (335)
Vismodegib Exerts Targeted Efficacy Against Recurrent Sonic Hedgehog-Subgroup Medulloblastoma: Results From Phase II Pediatric Brain Tumor Consortium Studies PBTC-025B and PBTC-032. (2015) (325)
Thyroid hormone receptor β is essential for development of auditory function (1996) (321)
Regulation of c-fos expression in transgenic mice requires multiple interdependent transcription control elements (1995) (318)
The product of a fos-related gene, fra-1, binds cooperatively to the AP-1 site with Jun: transcription factor AP-1 is comprised of multiple protein complexes. (1989) (292)
Induction of c-fos during myelomonocytic differentiation and macrophage proliferation (1985) (292)
Structure of the FBJ murine osteosarcoma virus genome: molecular cloning of its associated helper virus and the cellular homolog of the v-fos gene from mouse and human cells (1983) (270)
Role of DNA 5-methylcytosine transferase in cell transformation by fos. (1999) (256)
Activation of AP-1 and of a nuclear redox factor, Ref-1, in the response of HT29 colon cancer cells to hypoxia. (1994) (255)
Role of reelin in the control of brain development 1 Published on the World Wide Web on 21 October 1997. 1 (1998) (255)
Proto-oncogene transcription factors and epilepsy. (1991) (252)
Fos-Jun heterodimers and jun homodimers bend DNA in opposite orientations: Implications for transcription factor cooperativity (1991) (239)
BGEM: An In Situ Hybridization Database of Gene Expression in the Embryonic and Adult Mouse Nervous System (2006) (236)
Loss of suppressor-of-fused function promotes tumorigenesis (2007) (233)
Immediate-early genes: ten years on (1995) (229)
Memories of fos (1987) (227)
The normal patched allele is expressed in medulloblastomas from mice with heterozygous germ-line mutation of patched. (2000) (225)
Stimulation and inhibition of growth by EGF in different A431 cell clones is accompanied by the rapid induction of c‐fos and c‐myc proto‐oncogenes. (1985) (222)
Fos and Jun repress transcription activation by NF-IL6 through association at the basic zipper region (1994) (221)
Viral and cellular fos proteins are complexed with a 39,000-dalton cellular protein (1985) (214)
A molecular fingerprint for medulloblastoma. (2003) (212)
Phase I Study of Vismodegib in Children with Recurrent or Refractory Medulloblastoma: A Pediatric Brain Tumor Consortium Study (2013) (209)
Direct cloning of leucine zipper proteins: Jun binds cooperatively to the CRE with CRE-BP1. (1990) (204)
Transient inhibition of the Hedgehog pathway in young mice causes permanent defects in bone structure. (2008) (202)
Expression and purification of the leucine zipper and DNA-binding domains of Fos and Jun: both Fos and Jun contact DNA directly. (1990) (200)
Transcriptional activation and repression by Fos are independent functions: the C terminus represses immediate-early gene expression via CArG elements (1990) (198)
The Fos protein complex is associated with DNA in isolated nuclei and binds to DNA cellulose. (1986) (197)
Reelin Regulates the Development and Synaptogenesis of the Layer-Specific Entorhino-Hippocampal Connections (1999) (197)
Disabled-1 Binds to the Cytoplasmic Domain of Amyloid Precursor-Like Protein 1 (1999) (193)
Rescue of Ataxia and Preplate Splitting by Ectopic Expression of Reelin in reeler Mice (2002) (185)
Maf and Nrl can bind to AP-1 sites and form heterodimers with Fos and Jun. (1994) (183)
Phosphorylation of c-Fos at the C-terminus enhances its transforming activity. (1996) (176)
ERBB receptor signaling promotes ependymoma cell proliferation and represents a potential novel therapeutic target for this disease. (2002) (176)
Calcium as a modulator of the immediate-early gene cascade in neurons. (1988) (175)
Removal of a 67-base-pair sequence in the noncoding region of protooncogene fos converts it to a transforming gene. (1985) (171)
Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro (1993) (171)
Transcriptional regulation by Fos and Jun in vitro: interaction among multiple activator and regulatory domains (1991) (168)
The Oncogene Handbook (1988) (164)
Hedgehog signaling regulates the generation of ameloblast progenitors in the continuously growing mouse incisor (2010) (164)
The tumor suppressors Ink4c and p53 collaborate independently with Patched to suppress medulloblastoma formation. (2005) (164)
Kainic acid-induced neuronal death is associated with DNA damage and a unique immediate-early gene response in c-fos-lacZ transgenic rats (1995) (163)
Candidate product of the FBJ murine osteosarcoma virus oncogene: characterization of a 55,000-dalton phosphoprotein (1982) (161)
Thyroid Hormone Regulates reelin and dab1Expression During Brain Development (1999) (159)
Mutant mice with scrambled brains: understanding the signaling pathways that control cell positioning in the CNS. (1999) (158)
Cerebellar Disorganization Characteristic of Reeler in Scrambler Mutant Mice Despite Presence of Reelin (1997) (158)
Thyroid hormone receptor beta is essential for development of auditory function. (1996) (157)
DNA bending by Fos and Jun: the flexible hinge model. (1991) (154)
Altered protein conformation on DNA binding by Fos and Jun (1990) (154)
Reeler: new tales on an old mutant mouse (1998) (153)
A conserved region adjacent to the basic domain is required for recognition of an extended DNA binding site by Maf/Nrl family proteins. (1994) (151)
Crk and Crk-Like Play Essential Overlapping Roles Downstream of Disabled-1 in the Reelin Pathway (2008) (148)
Shh pathway activity is down-regulated in cultured medulloblastoma cells: implications for preclinical studies. (2006) (148)
Selective DNA bending by a variety of bZIP proteins (1993) (144)
The human reelin gene: isolation, sequencing, and mapping on chromosome 7. (1997) (144)
Gli1 is important for medulloblastoma formation in Ptc1+/− mice (2005) (142)
Targeting medulloblastoma: small-molecule inhibitors of the Sonic Hedgehog pathway as potential cancer therapeutics. (2005) (141)
Mouse embryos cloned from brain tumors. (2003) (139)
Identification of Reelin-induced Sites of Tyrosyl Phosphorylation on Disabled 1* (2001) (139)
Deletion of Shp2 in the Brain Leads to Defective Proliferation and Differentiation in Neural Stem Cells and Early Postnatal Lethality (2007) (134)
The Reelin Pathway Modulates the Structure and Function of Retinal Synaptic Circuitry (2001) (126)
Induction of c‐fos mRNA Expression by Afterdischarge in the Hippocampus of Naive and Kindled Rats (1990) (124)
Crystal Structures of the Dab Homology Domains of Mouse Disabled 1 and 2* (2003) (122)
Transcription factor interactions: basics on zippers (1991) (119)
Patched2 modulates tumorigenesis in patched1 heterozygous mice. (2006) (119)
Analysis of Fos protein complexes and Fos-related antigens by high-resolution two-dimensional gel electrophoresis. (1987) (119)
Escape from redox regulation enhances the transforming activity of Fos. (1993) (118)
The hedgehog pathway and neurological disorders. (2006) (114)
Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease. (2012) (109)
Microinjection of transforming ras protein induces c-fos expression (1987) (109)
Binding of purified Reelin to ApoER2 and VLDLR mediates tyrosine phosphorylation of Disabled-1. (2003) (109)
Cyclin-Dependent Kinase 5 Phosphorylates Disabled 1 Independently of Reelin Signaling (2002) (107)
Encounters with Fos and Jun on the road to AP-1. (1990) (105)
Dok-7 regulates neuromuscular synapse formation by recruiting Crk and Crk-L. (2010) (101)
Cell transformation by c-fos requires an extended period of expression and is independent of the cell cycle (1994) (101)
Disabled‐1 is expressed in type AII amacrine cells in the mouse retina (2000) (100)
Jun is phosphorylated by several protein kinases at the same sites that are modified in serum-stimulated fibroblasts (1992) (100)
fos-lacZ transgenic mice: mapping sites of gene induction in the central nervous system. (1992) (99)
Barium modulates c-fos expression and post-translational modification. (1986) (95)
Thyrotropin Regulation by Thyroid Hormone in Thyroid Hormone Receptor β-Deficient Mice. (1997) (95)
Detection of the reelin breakpoint in reeler mice. (1996) (93)
Transient induction of c-fos and c-myc in an immediate consequence of growth factor stimulation. (1985) (91)
FBR murine osteosarcoma virus. II. Nucleotide sequence of the provirus reveals that the genome contains sequences acquired from two cellular genes. (1984) (91)
Identification of a 39,000-dalton protein in cells transformed by the FBJ murine osteosarcoma virus. (1982) (90)
Activation of the transforming potential of the human fos proto-oncogene requires message stabilization and results in increased amounts of partially modified fos protein (1988) (90)
Cardiovascular and Craniofacial Defects in Crk-Null Mice (2006) (89)
Redox regulation of AP-1: a link between transcription factor signaling and DNA repair. (1996) (86)
Regulation of a fos-lacZ fusion gene: a paradigm for quantitative analysis of stimulus-transcription coupling. (1991) (86)
Menin epigenetically represses Hedgehog signaling in MEN1 tumor syndrome. (2013) (85)
FBR murine osteosarcoma virus. I. Molecular analysis and characterization of a 75,000-Da gag-fos fusion product. (1984) (85)
Fos and jun cooperate in transcriptional regulation via heterologous activation domains (1990) (84)
Dysfunctions in Mice by NMDA Receptor Point Mutations NR1(N598Q) and NR1(N598R) (2000) (80)
The structure and function of the fos proto-oncogene. (1989) (78)
Expression of c‐fos in NIH3T3 cells is very low but inducible throughout the cell cycle. (1986) (77)
Reovirus prolongs survival and reduces the frequency of spinal and leptomeningeal metastases from medulloblastoma. (2003) (76)
Transcription repression in oncogenic transformation: common targets of epigenetic repression in cells transformed by Fos, Ras or Dnmt1 (2004) (74)
Extended life span and tumorigenicity of nonestablished mouse connective tissue cells transformed by the fos oncogene of FBR-MuSV (1985) (71)
Tyrosine phosphorylated Disabled 1 recruits Crk family adapter proteins. (2004) (71)
Inducible proto-oncogenes of the nervous system: their contribution to transcription factors and neuroplasticity. (1990) (69)
Fos‐Like Immunoreactivity Induced by Seizure in Mice Is Specifically Associated With Euchromatin in Neurons (1989) (66)
Energy transfer analysis of Fos-Jun dimerization and DNA binding. (1994) (66)
Fos is phosphorylated by p34cdc2, cAMP-dependent protein kinase and protein kinase C at multiple sites clustered within regulatory regions. (1991) (66)
Differential binding of ligands to the apolipoprotein E receptor 2. (2003) (65)
Cysteine 64 of Ref-1 Is Not Essential for Redox Regulation of AP-1 DNA Binding (2003) (64)
Dimerization and DNA binding alter phosphorylation of Fos and Jun. (1993) (64)
Glucocorticoid compounds modify smoothened localization and hedgehog pathway activity. (2012) (62)
Reelin mRNA expression during embryonic brain development in the chick (2000) (62)
Analysis of dimerization and DNA binding functions in Fos and Jun by domain-swapping: involvement of residues outside the leucine zipper/basic region. (1990) (60)
A ubiquitous nuclear protein stimulates the DNA-binding activity of fos and jun indirectly. (1990) (58)
Isolation of an allele of reeler by insertional mutagenesis. (1994) (57)
Zen and the art of Fos and Jun (1995) (57)
Disabled-1 interacts with a novel developmentally regulated protocadherin. (2001) (56)
Developmental expression of thyroid hormone receptor &bgr;2 protein in cone photoreceptors in the mouse (2009) (55)
Ubiquitinylation of Transcription Factors c-Jun and c-Fos Using Reconstituted Ubiquitinylating Enzymes (*) (1996) (54)
Redox Regulation of Ap-1 (1996) (53)
Functional NMDA Receptors Are Transiently Active and Support the Survival of Purkinje Cells in Culture (1996) (52)
Calcium and Proto‐Oncogene Involvement in the Immediate‐Early Response in the Nervous System (1989) (49)
The transcription activation domains of Fos and Jun induce DNA bending through electrostatic interactions (1997) (48)
Design of a "minimAl" homeodomain: the N-terminal arm modulates DNA binding affinity and stabilizes homeodomain structure. (1994) (47)
Inducible proto-oncogene transcription factors: third messengers in the brain? (1990) (45)
Thyrotropin regulation by thyroid hormone in thyroid hormone receptor beta-deficient mice. (1997) (45)
Cortical development: Cdk5 gets into sticky situations (2000) (45)
CRK proteins selectively regulate T cell migration into inflamed tissues. (2015) (41)
Inhibition of jun transformation by a mutated fos gene: design of an anti-oncogene. (1991) (41)
A central role for Fos in human B- and T-cell NFAT (nuclear factor of activated T cells): an acidic region is required for in vitro assembly (1994) (40)
Methylation matters: modeling a manageable genome. (2002) (40)
c‐Jun stimulates origin‐dependent DNA unwinding by polyomavirus large Tantigen. (1996) (40)
Medulloblastomas derived from Cxcr6 mutant mice respond to treatment with a smoothened inhibitor. (2007) (39)
The fos gene product undergoes extensive post-translational modification in eukaryotic but not in prokaryotic cells. (1986) (39)
Statins Synergize with Hedgehog Pathway Inhibitors for Treatment of Medulloblastoma (2018) (38)
Selective Activation of Calcium Permeability by Aspartate in Purkinje Cells (1996) (38)
Temporal and spatial expression of a fos-lacZ transgene in the developing nervous system. (1992) (38)
Crk1/2 and CrkL form a hetero-oligomer and functionally complement each other during podocyte morphogenesis (2014) (38)
CHAPTER 14 - Fos and Jun: intermediary transcription factors (1991) (37)
Crk proteins transduce FGF signaling to promote lens fiber cell elongation (2018) (37)
Fos-Jun dimerization promotes interaction of the basic region with TFIIE-34 and TFIIF (1996) (36)
Astrocytes Promote Medulloblastoma Progression through Hedgehog Secretion. (2017) (35)
Interaction of Disabled-1 and the GTPase activating protein Dab2IP in mouse brain. (2003) (35)
Genetic analysis of thyroid hormone receptors in development and disease. (1996) (35)
Brain development: Integrins and the Reelin pathway (2001) (34)
Beyond the second messenger: oncogenes and transcription factors. (1988) (33)
N-terminal variants of thyroid hormone receptor beta: differential function and potential contribution to syndrome of resistance to thyroid hormone. (1995) (33)
Apoptosis in the nervous system: new revelations. (1995) (33)
Review : The Immediate-Early Gene Response and Neuronal Death and Regeneration (1995) (32)
Generation of a mouse model of atypical teratoid/rhabdoid tumor of the central nervous system through combined deletion of Snf5 and p53. (2015) (31)
Fos and Jun: oncogenic transcription factors. (1992) (29)
Essential roles of Crk and CrkL in fibroblast structure and motility (2014) (28)
30 Dangerous Liaisons: Fos and Jun, Oncogenic Transcription Factors (1992) (28)
Audiogenic seizure susceptibility in thyroid hormone receptor β-deficient mice (2001) (28)
Deletion of the gag region from FBR murine osteosarcoma virus does not affect its enhanced transforming activity (1985) (28)
A fos-lac Z transgenic mouse that can be used for neuroanatomic mapping. (1993) (28)
Analysis of c-Fos and c-Jun redox-dependent DNA binding activity. (1994) (27)
Glutamate, Immediate‐Early Genes, and Cell Death in the Nervous System (1993) (27)
Regulation of proto-oncogenes in rat parotid acinar cells in vitro after stimulation of beta-adrenergic receptors. (1988) (27)
Continuous c-fos expression precedes programmed cell death in vivo (1993) (27)
Components of the Reelin signaling pathway are expressed in the spinal cord (2004) (26)
Differential roles for Fos and Jun in DNA-binding: redox-dependent and independent functions. (1993) (26)
Cancer: Hedgehog's other great trick (2008) (26)
Absence of thyroid hormone receptor β–retinoid X receptor interactions in auditory function and in the pituitary–thyroid axis (1998) (25)
Reproducibility of academic preclinical translational research: lessons from the development of Hedgehog pathway inhibitors to treat cancer (2018) (24)
The Ketogenic Diet Does Not Affect Growth of Hedgehog Pathway Medulloblastoma in Mice (2015) (24)
Nestin Mediates Hedgehog Pathway Tumorigenesis. (2016) (23)
Regulation of proto-oncogene expression in adult and developing lungs (1993) (22)
A transcriptome map of cellular transformation by the fos oncogene (2005) (22)
Crossed signals: oncogenic transcription factors (1992) (22)
Ectopic expression of reelin alters migration of sympathetic preganglionic neurons in the spinal cord (2009) (21)
Gene expression profiles of mouse retinas during the second and third postnatal weeks (2006) (21)
A phase I pharmacokinetic trial of sonic hedgehog (SHH) antagonist GDC-0449 in pediatric patients with recurrent or refractory medulloblastoma: A Pediatric Brain Tumor Consortium study (PBTC 25). (2010) (19)
The effects of phorbol ester and Ca ionophore on c-fos and c-myc expression and on DNA synthesis in human lymphocytes are not directly related. (1987) (18)
Gene dosage in mice—BAC to the future (1999) (18)
Transcriptional Regulation byFosandJunInVitro: Interaction among Multiple Activator andRegulatory Domains (1991) (16)
The regulation of c-fos: too much is never enough. (1993) (16)
Design of an autonomous intelligent Demand-Side Management system using stochastic optimisation evolutionary algorithms (2015) (16)
The carboxy terminus of the viral Jun oncoprotein is required for complex formation with the cellular Fos protein. (1989) (16)
Macrophages in SHH subgroup medulloblastoma display dynamic heterogeneity that varies with treatment modality. (2021) (15)
A gene expression approach to mapping the functional maturation of the hippocampus. (1998) (15)
Spontaneous and evoked glutamate signalling influences Fos-lacZ expression and pyramidal cell death in hippocampal slice cultures from transgenic rats. (1995) (15)
Developmental mouse brain gene expression maps (2006) (15)
Fosvergnügen The Excitement of Immediate‐Early Genes (1991) (14)
PTHrP Treatment Fails to Rescue Bone Defects Caused by Hedgehog Pathway Inhibition in Young Mice (2011) (14)
Loss of p 53 but not ARF Accelerates Medulloblastoma in Mice Heterozygous for patched 1 (2001) (14)
Audiogenic seizure susceptibility in thyroid hormone receptor beta-deficient mice. (2001) (14)
Smart transcription factors (1995) (13)
Transcription Factors as Molecular Mediators in Cell Death (1994) (13)
Genomic answers for children: Dynamic analyses of >1000 pediatric rare disease genomes (2021) (13)
Fibroblast Growth Requires CT10 Regulator of Kinase (Crk) and Crk-like (CrkL)* (2016) (13)
A neurogenomics approach to gene expression analysis in the developing brain. (2004) (13)
The adaptor protein CRK is a pro-apoptotic transducer of endoplasmic reticulum stress. (2016) (13)
Developmental expression of Fos-lacZ in the brains of postnatal transgenic rats. (1996) (12)
A prospective phase II study to determine the efficacy of GDC 0449 (vismodegib) in adults with recurrent medulloblastoma (MB): A Pediatric Brain Tumor Consortium study (PBTC 25B). (2013) (11)
Viral andCellular fosProteins AreComplexed witha39,000-Dalton Cellular Protein (1985) (10)
Levels of fos, ets2, and myb proto-oncogene RNAs correlate with segregation of chromosome 11 of normal cells and with suppression of tumorigenicity in human cell hybrids (1987) (10)
Automatic Categorization of Statute Documents (1997) (10)
E-democracy as the future face of democracy: a case study of the 2011 Irish elections (2011) (9)
Use of fluorescence resonance energy transfer to estimate intramolecular distances in the Msx-1 homeodomain. (1995) (8)
mPPP1R16B is a novel mouse protein phosphatase 1 targeting subunit whose mRNA is located in cell bodies and dendrites of neurons in four distinct regions of the brain. (2002) (8)
DNA Bending by Fos and Jun: Structural and Functional Implications (1993) (8)
A call to action: Issuing a diversity and inclusion challenge to research organizations (2021) (8)
Leukotriene Synthesis Is Critical for Medulloblastoma Progression (2019) (8)
Yayoi era mutation disrupts brain and muscle (1998) (8)
Proto-oncogene CT10-regulated kinase (CRK) is a pro-apoptotic transducer of endoplasmic reticulum stress (2011) (7)
Dendritic planarity of Purkinje cells is independent of Reelin signaling (2014) (7)
Cortical development in the presenilin‐1 null mutant mouse fails after splitting of the preplate and is not due to a failure of reelin‐dependent signaling (2008) (7)
Whole-exome identifies RXRG and TH germline variants in familial isolated prolactinoma. (2016) (6)
Creating an Intensive Care Unit From a Postanesthesia Care Unit for the COVID-19 Surge at the Veterans Affairs Ann Arbor Healthcare System. (2020) (6)
Cellular Immediate-Early Genes in the Nervous System: Genes for All Reasons? (1994) (5)
Mouse models and mouse supermodels (2010) (5)
Immediate early genes: their involvement in physiological and pathological responses in the nervous system. (1993) (5)
Who was Responsible for the Dardanelles Naval Fiasco (2011) (4)
Alternative Splicing Disabled by Nova2 (2010) (4)
Tumor Syndrome Menin Epigenetically Represses Hedgehog Signaling in MEN 1 (2013) (4)
Statins repress hedgehog signaling in medulloblastoma with no bone toxicities (2021) (4)
Reeler gene discrepancies (1995) (4)
Crk and CrkL are required for cell transformation by v‐fos and v‐ras (2016) (4)
Lost in Translation: The Future of Cancer Research? (2005) (4)
The Grand Deception: Churchill and the Dardanelles (2015) (3)
Two faces of evil : cancer and neurodegeneration (2011) (3)
Requirement for Crk and CrkL during postnatal lens development. (2020) (3)
CELL BIOLOGY AND SIGNALING (2010) (2)
Reply to the Letter to the Editor by Diamandis (2006) (1)
The deconstruction of Simpson, the man with the donkey (1996) (1)
Erratum: Radial glia cells are candidate stem cells of ependymoma (Cancer Cell (October 2005) 8 (323-335)) (2006) (1)
Single-cell transcriptomic profile reveals macrophage heterogeneity in medulloblastoma and their treatment-dependent recruitment (2020) (1)
Neurobiology: Reelin Mediates Form and Function (2014) (1)
Quantitative assessment of glioblastoma phenotypes in vitro establishes cell migration as a robust readout of Crk and CrkL activity (2021) (1)
Viral and Cellular Fos Gene (1985) (1)
The Chemung County Perinatal Dental Coalition. (2009) (1)
c-fos and Signal Transduction In Vivo. (1998) (0)
Structure oftheFBJMurineOsteosarcoma Virus Genome: Molecular Cloning ofItsAssociated Helper Virus andthe Cellular Homologofthev-fos GenefromMouseandHuman Cells (1983) (0)
A Central RoleforFosinHumanB-andT-Cell NFAT (Nuclear Factor ofActivated TCells): anAcidic Region IsRequired forInVitro Assembly (1994) (0)
Abstract 5423: Identification of immunotargets in Hedgehog medulloblastoma for immunotherapy (2015) (0)
FosIsa Preferential Target ofGlucocorticoid Receptor Inhibition ofAP-1Activity InVitro (1993) (0)
Regulation of c-fos Expression by Voltage-Dependent Calcium Channels (1989) (0)
Abstract C110: Abstract B: Hedgehog pathway inhibition in young mice results in severe bone defects (2009) (0)
Molecular oncology today. Oncoigenes and viral genes. 1984. Edited by g. F. V. WOUDE, A. J. LEVINE, W. C. TOPP and J. D. WATSON. Cancer Cells, Volume 2. Pp. 564. Cold Spring Harbor Laboratory. $65 ($78 outside US) (1985) (0)
region.through association at the basic zipper Fos and Jun repress transcription activation by (2013) (0)
Crystal structure of the phosphotyrosine binding domain(PTB) of mouse Disabled 2(Dab2):implications for Reeling signaling (2003) (0)
FosandJunRepress Transcription Activation byNF-IL6 through Association attheBasicZipper Region (1994) (0)
UNIVERSIDADE FEDERAL DE MINAS GERAIS Graduate Program in Molecular Medicine (2017) (0)
Cooperative Interactions of Transcription Factors at the DNA Structure Designated an Ap-1 Site (1989) (0)
fos and the Immediate-Early Response in the Central Nervous System (2020) (0)
From Bings to Biology: Taking a Different Path in Cancer Research (2003) (0)
Induction of C-FOS, C-MYC and P53 by US -adrenergic receptor (US -AR) stimulation of rat parotid acinar cells (RPAC) (1987) (0)
Shh pathway activity is maintained in direct allografts of medulloblastoma cells whereas it is irreversibly lost in cell culture: Implications for preclinical studies of smoothened antagonists (2006) (0)
Activation of AP-1 and of a nuclear redox factor, Ref-1, in the response of HT29 colon cancer cells to hypoxia (1994) (0)
Targeting Children’s Brain Tumors: Development of Hedgehog Pathway Inhibitors for Medulloblastoma (2011) (0)
Fos and Jun: Oncogenic Immediate-early Tran- Scription Factors Transcrip- Tional Regulation by Fos and Jun in Vitro : Interaction (0)
[Stimulation of human lymphocytes by phorbol acetate and a calcium ionophore induces an early transient expression of the c-fos oncogene followed by c-myc]. (1986) (0)
JunIsPhosphorylated bySeveral Protein Kinases attheSameSites ThatAreModified inSerum-Stimulated Fibroblasts (1992) (0)
Dendritic planarity of Purkinje cells is independent of Reelin signaling (2014) (0)
Activation oftheTransforming Potential oftheHumanfos Proto-Oncogene Requires MessageStabilization andResults in Increased AmountsofPartially Modified fosProtein (1988) (0)
Levels offos,ets2, andmybProto-Oncogene RNAsCorrelate with Segregation ofChromosome 11ofNormalCells andwith Suppression ofTumorigenicity inHumanCellHybrids (1987) (0)
Mouse Embryos Cloned from Brain Tumors 1 (2003) (0)
Advances in Brief Mouse Embryos Cloned from Brain Tumors 1 (2003) (0)
Crystal structure of the phosphotyrosine binding domain (PTB) of mouse Disabled 1 (Dab1) (2003) (0)
IMMU-19. IMMUNE RESPONSE OF MOUSE HEDGEHOG MEDULLOBLASTOMA TREATED WITH SMOOTHENED INHIBITOR (2017) (0)
Transgenic mice studies of immediate-early genes: from markers to mutants (1995) (0)
Methylation Matters : Modeling a Manageable Genome 1 (2002) (0)
proteins.DNA bending by a variety of bZIP (2013) (0)
Longboats, Oak, and the Dark Days of the Northmen: Seamus Heaney’s Barbarisms in The Secret of Kells (2017) (0)
Zombie Myths of Australian Military History. Edited by Craig Stockings (2010) (0)
CRK/CRKL proteins regulate T cell migration and activation (P5104) (2013) (0)
Human genetics. Smart transcription factors. (1995) (0)
Opporfunifies Will There Be For Government Girls After the War (2010) (0)
The Peter Gellhorn Edition: General Statement and 16 associated scores (2016) (0)
Optimising Charging of Electric Vehicles through the use of Genetic Algorithms (2014) (0)
Transforming the former Waterloo International Terminal for commuter services (2017) (0)
Anzac Legacies: Australians and the aftermath of war (2010) (0)
506 SAME DAY DISCHARGE IS SAFE IN SELECTED PATIENTS FOLLOWING LAPAROSCOPIC RIGHT COLECTOMY IN THE NATIONAL SURGICAL QUALITY IMPROVEMENT PROJECT (2020) (0)
Q&A: Tom Curran on translational research. (2012) (0)
The Problem of Relations: A Rejoinder to Bradley (1965) (0)

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