Yosef Shiloh

Q: What Schools Are Affiliated With Yosef Shiloh

Yosef Shiloh is affiliated with the following schools: University of California, Berkeley, University of Queensland, Weizmann Institute of Science, California Institute of Technology, Tel Aviv University, Stanford University, Massachusetts General Hospital, Harvard University

Yosef Shiloh's AcademicInfluence.com Rankings

Yosef Shiloh

Biology

#7078

World Rank

#9937

Historical Rank

Cell Biology

#252

World Rank

#260

Historical Rank

Genetics

#669

World Rank

#755

Historical Rank

Molecular Biology

#781

World Rank

#801

Historical Rank

biology Degrees

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Biology

Yosef Shiloh's Degrees

PhD Molecular Biology University of California, Berkeley
Masters Biochemistry Stanford University

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Yosef Shiloh's Published Works

Number of citations in a given year to any of this author's works

Total number of citations to an author for the works they published in a given year. This highlights publication of the most important work(s) by the author

Published Works

ATM and ATR Substrate Analysis Reveals Extensive Protein Networks Responsive to DNA Damage (2007) (3001)
A single ataxia telangiectasia gene with a product similar to PI-3 kinase. (1995) (2772)
ATM and related protein kinases: safeguarding genome integrity (2003) (2675)
Atm-Deficient Mice: A Paradigm of Ataxia Telangiectasia (1996) (1485)
Enhanced phosphorylation of p53 by ATM in response to DNA damage. (1998) (1396)
Requirement of the MRN complex for ATM activation by DNA damage (2003) (1104)
Ataxia telangiectasia-mutated phosphorylates Chk2 in vivo and in vitro. (2000) (871)
ATM signaling facilitates repair of DNA double-strand breaks associated with heterochromatin. (2008) (869)
The ATM protein kinase: regulating the cellular response to genotoxic stress, and more. (2013) (794)
ATM-dependent phosphorylation of Mdm2 on serine 395: role in p53 activation by DNA damage. (2001) (782)
Chromatin relaxation in response to DNA double-strand breaks is modulated by a novel ATM- and KAP-1 dependent pathway (2006) (716)
The ATM protein kinase: regulating the cellular response to genotoxic stress, and more (2013) (677)
The genetic defect in ataxia-telangiectasia. (1997) (647)
ATM and ATR: networking cellular responses to DNA damage. (2001) (636)
The ATM-mediated DNA-damage response: taking shape. (2006) (576)
The complete sequence of the coding region of the ATM gene reveals similarity to cell cycle regulators in different species. (1995) (542)
Ataxia-telangiectasia and the Nijmegen breakage syndrome: related disorders but genes apart. (1997) (519)
Functional link between ataxia-telangiectasia and Nijmegen breakage syndrome gene products (2000) (509)
Interaction between ATM protein and c-Abl in response to DNA damage (1997) (489)
Rapid ATM-dependent phosphorylation of MDM2 precedes p53 accumulation in response to DNA damage. (1999) (487)
TEL1, an S. cerevisiae homolog of the human gene mutated in ataxia telangiectasia, is functionally related to the yeast checkpoint gene MEC1 (1995) (368)
ATM: from gene to function. (1998) (358)
ATM deficiency and oxidative stress: a new dimension of defective response to DNA damage. (2002) (356)
Requirement of ATM-dependent monoubiquitylation of histone H2B for timely repair of DNA double-strand breaks. (2011) (356)
EXPANDER – an integrative program suite for microarray data analysis (2005) (347)
Functional link of BRCA1 and ataxia telangiectasia gene product in DNA damage response (2000) (337)
Genome-wide in silico identification of transcriptional regulators controlling the cell cycle in human cells. (2003) (336)
ATM: genome stability, neuronal development, and cancer cross paths. (2001) (312)
Jun NH2-Terminal Kinase Phosphorylation of p53 on Thr-81 Is Important for p53 Stabilization and Transcriptional Activities in Response to Stress (2001) (310)
Ataxia Telangiectasia Mutated (ATM) Is Essential for DNA-PKcs Phosphorylations at the Thr-2609 Cluster upon DNA Double Strand Break* (2007) (294)
Nuclear retention of ATM at sites of DNA double strand breaks. (2001) (293)
ATM: A mediator of multiple responses to genotoxic stress (1999) (292)
ATM-Dependent and -Independent Dynamics of the Nuclear Phosphoproteome After DNA Damage (2010) (277)
Predominance of null mutations in ataxia-telangiectasia. (1996) (276)
The histone H2B-specific ubiquitin ligase RNF20/hBRE1 acts as a putative tumor suppressor through selective regulation of gene expression. (2008) (263)
Recombinant ATM protein complements the cellular A-T phenotype (1997) (261)
Genotype-phenotype relationships in ataxia-telangiectasia and variants. (1998) (240)
Increased oxidative stress in ataxia telangiectasia evidenced by alterations in redox state of brains from Atm-deficient mice. (2001) (225)
Beyond ATM: The protein kinase landscape of the DNA damage response (2011) (203)
Involvement of Matrin 3 and SFPQ/NONO in the DNA damage response (2010) (193)
Phosphorylation of Hdmx mediates its Hdm2- and ATM-dependent degradation in response to DNA damage. (2005) (192)
Expander: from expression microarrays to networks and functions (2010) (189)
ATM Is Required for IκB Kinase (IKK) Activation in Response to DNA Double Strand Breaks* (2000) (175)
Genomic Organization of the ATM gene. (1996) (173)
ATM-Mediated Phosphorylations Inhibit Mdmx/Mdm2 Stabilization by HAUSP in Favor of p53 Activation (2005) (168)
Fragments of ATM which have dominant-negative or complementing activity (1997) (167)
ATM-dependent phosphorylation of ATF2 is required for the DNA damage response. (2005) (166)
Ataxia-telangiectasia: is ATM a sensor of oxidative damage and stress? (1997) (164)
The ataxia-telangiectasia gene product, a constitutively expressed nuclear protein that is not up-regulated following genome damage. (1997) (164)
ATM binds to β-adaptin in cytoplasmic vesicles (1998) (163)
Ataxia-Telangiectasia: Closer to Unraveling the Mystery (1995) (158)
Sporadic amplification of the HER2/neu protooncogene in adenocarcinomas of various tissues. (1988) (153)
Inhibition of transforming growth factor-beta1 signaling attenuates ataxia telangiectasia mutated activity in response to genotoxic stress. (2006) (151)
Ataxia-telangiectasia: mutations in ATM cDNA detected by protein-truncation screening. (1996) (148)
Definition and refinement of chromosome 11 regions of loss of heterozygosity in breast cancer: identification of a new region at 11q23.3. (1995) (146)
The neurological phenotype of ataxia-telangiectasia: solving a persistent puzzle. (2008) (143)
Downregulation of the type 1 insulin-like growth factor receptor in mouse melanoma cells is associated with enhanced radiosensitivity and impaired activation of Atm kinase (2001) (134)
The role of the DNA damage response in neuronal development, organization and maintenance. (2008) (133)
Matrin 3 Binds and Stabilizes mRNA (2011) (132)
DNA Damage-Induced Phosphorylation of MdmX at Serine 367 Activates p53 by Targeting MdmX for Mdm2-Dependent Degradation (2005) (130)
Optimal function of the DNA repair enzyme TDP1 requires its phosphorylation by ATM and/or DNA‐PK (2009) (129)
Ionizing radiation induces ataxia telangiectasia mutated kinase (ATM)-mediated phosphorylation of LKB1/STK11 at Thr-366. (2002) (129)
Ataxia-telangiectasia locus: sequence analysis of 184 kb of human genomic DNA containing the entire ATM gene. (1997) (121)
Programs for Cell Death: Apoptosis is Only One Way to Go (2007) (117)
Abnormal response of ataxia-telangiectasia cells to agents that break the deoxyribose moiety of DNA via a targeted free radical mechanism. (1983) (115)
Accelerated carcinogenesis following liver regeneration is associated with chronic inflammation-induced double-strand DNA breaks (2010) (112)
Ataxia-telangiectasia: founder effect among north African Jews. (1996) (101)
ATM: expanding roles as a chief guardian of genome stability. (2014) (99)
Differential Roles of ATM- and Chk2-Mediated Phosphorylations of Hdmx in Response to DNA Damage (2006) (99)
Selective loss of dopaminergic nigro-striatal neurons in brains of Atm-deficient mice. (1998) (97)
Systemic DNA damage responses in aging and diseases. (2016) (94)
Dissection of a DNA-damage-induced transcriptional network using a combination of microarrays, RNA interference and computational promoter analysis (2005) (91)
Accumulation of DNA Damage and Reduced Levels of Nicotine Adenine Dinucleotide in the Brains of Atm-deficient Mice* (2002) (90)
Transcriptional modulation induced by ionizing radiation: p53 remains a central player (2011) (90)
Relationship of the ataxia-telangiectasia protein ATM to phosphoinositide 3-kinase. (1995) (88)
UBQLN4 Represses Homologous Recombination and Is Overexpressed in Aggressive Tumors (2019) (88)
Ataxia-telangiectasia (A-T): An emerging dimension of premature ageing (2017) (86)
WRN is required for ATM activation and the S-phase checkpoint in response to interstrand cross-link-induced DNA double-strand breaks. (2008) (83)
The ATM gene and protein: possible roles in genome surveillance, checkpoint controls and cellular defence against oxidative stress. (1997) (80)
SPIKE: a database of highly curated human signaling pathways (2010) (78)
Parallel induction of ATM-dependent pro- and antiapoptotic signals in response to ionizing radiation in murine lymphoid tissue (2006) (77)
Nuclear Ataxia-Telangiectasia Mutated (ATM) Mediates the Cellular Response to DNA Double Strand Breaks in Human Neuron-like Cells* (2006) (75)
Loss of heterozygosity at 11q23.1 in breast carcinomas: Indication for involvement of a gene distal and close to ATM (1997) (72)
ATM binds to beta-adaptin in cytoplasmic vesicles. (1998) (71)
Ataxia-telangiectasia: a multifaceted genetic disorder associated with defective signal transduction. (1996) (71)
Ataxia-telangiectasia: structural diversity of untranslated sequences suggests complex post-transcriptional regulation of ATM gene expression. (1997) (70)
ATM (ataxia telangiectasia mutated): expanding roles in the DNA damage response and cellular homeostasis. (2001) (70)
A single origin of phenylketonuria in Yemenite Jews (1990) (69)
Atm knock-in mice harboring an in-frame deletion corresponding to the human ATM 7636del9 common mutation exhibit a variant phenotype. (2001) (68)
Ataxia-telangiectasia: chronic activation of damage-responsive functions is reduced by α-lipoic acid (2001) (67)
Citrate boosts the performance of phosphopeptide analysis by UPLC-ESI-MS/MS. (2009) (67)
SPIKE – a database, visualization and analysis tool of cellular signaling pathways (2008) (67)
RNF20–RNF40: A ubiquitin‐driven link between gene expression and the DNA damage response (2011) (67)
Differential amplification, assembly, and relocation of multiple DNA sequences in human neuroblastomas and neuroblastoma cell lines. (1985) (66)
Corrigendum: The histone H2B-specific ubiquitin ligase RNF20/hBRE1 acts as a putative tumor suppressor through selective regulation of gene expression. (2008) (66)
The response of ataxia-telangiectasia homozygous and heterozygous skin fibroblasts to neocarzinostatin. (1982) (66)
Involvement of the nuclear proteasome activator PA28γ in the cellular response to DNA double-strand breaks (2011) (65)
Localization of an ataxia-telangiectasia gene to an ∼500-kb interval on chromosome 11q23.1 : linkage analysis of 176 families by an international consortium (1991) (65)
ATM‐mediated phosphorylation of polynucleotide kinase/phosphatase is required for effective DNA double‐strand break repair (2011) (59)
A human gene (DDX10) encoding a putative DEAD-box RNA helicase at 11q22-q23. (1996) (55)
Targeting double-strand breaks to replicating DNA identifies a subpathway of DSB repair that is defective in ataxia-telangiectasia cells. (1999) (54)
Amplification and rearrangement of DNA sequences from the chromosomal region 2p24 in human neuroblastomas. (1986) (53)
Identification and chromosomal localization of Atm, the mouse homolog of the ataxia-telangiectasia gene. (1996) (52)
ATM: Ready, Set, Go (2003) (51)
The Activation Of ATM Depends On Chromatin Interactions Occurring Prior To DNA-Damage Induction (2008) (51)
Parallel Profiling of the Transcriptome, Cistrome, and Epigenome in the Cellular Response to Ionizing Radiation (2014) (50)
Ataxia‐telangiectasia: Mild neurological presentation despite null ATM mutation and severe cellular phenotype (2007) (50)
Deciphering Transcriptional Regulatory Elements That Encode Specific Cell-Cycle Phasing by Comparative Genomics Analysis (2005) (48)
Colony-forming ability of ataxia-telangiectasia skin fibroblasts is an indicator of their early senescence and increased demand for growth factors. (1982) (48)
Induction and repair of DNA damage in normal and ataxia-telangiectasia skin fibroblasts treated with neocarzinostatin. (1983) (47)
Enhanced phosphorylation of p53 serine 18 following DNA damage in DNA-dependent protein kinase catalytic subunit-deficient cells. (1999) (46)
Cellular and molecular characteristics of an immortalized ataxia-telangiectasia (group AB) cell line. (1989) (46)
Cellular hypersensitivity to neocarzinostatin in ataxia-telangiectasia skin fibroblasts. (1982) (46)
ATM-mediated response to DNA double strand breaks in human neurons derived from stem cells. (2007) (45)
Detection of ataxia telangiectasia heterozygous cell lines by postirradiation cumulative labeling index: measurements with coded samples. (1987) (45)
The ATC (ataxia-telangiectasia complementation group C) locus localizes to 11q22-q23. (1991) (43)
Contribution of the Atm Protein to Maintaining Cellular Homeostasis Evidenced by Continuous Activation of the AP-1 Pathway in Atm-deficient Brains* (2003) (42)
Analysis of the Ataxia Telangiectasia Mutated-Mediated DNA Damage Response in Murine Cerebellar Neurons (2006) (41)
Genetic mapping of X-linked albinism-deafness syndrome (ADFN) to Xq26.3-q27.I. (1990) (41)
DNA amplification in human gastric carcinomas. (1993) (40)
Ataxia-telangiectasia and the ATM gene: Linking neurodegeneration, immunodeficiency, and cancer to cell cycle checkpoints (1996) (38)
New hallmarks of ageing: a 2022 Copenhagen ageing meeting summary (2022) (38)
Kinetics of O6-methylguanine repair in human normal and ataxia telangiectasia cell lines and correlation of repair capacity with cellular sensitivity to methylating agents. (1981) (37)
The Ubiquitin E3/E4 Ligase UBE4A Adjusts Protein Ubiquitylation and Accumulation at Sites of DNA Damage, Facilitating Double-Strand Break Repair. (2018) (37)
Amplification of c‐myc and c‐erbB‐2 proto‐oncogenes in human solid tumors: Frequency and clinical significance (1989) (36)
MRI evidence of white matter damage in a mouse model of Nijmegen breakage syndrome (2008) (33)
Functional genomic delineation of TLR-induced transcriptional networks (2007) (33)
Relatively low proportion of dystrophin gene deletions in Israeli Duchenne and Becker muscular dystrophy patients. (1994) (32)
Compound heterozygosity in nonphenylketonuria hyperphenylalanemia: the contribution of mutations for classical phenylketonuria. (1991) (32)
PIVOT: Protein Interacions VisualizatiOn Tool (2004) (32)
Detection of amplified dna sequences in human tumor cell lines by fluorescence in situ hybridization (1992) (32)
ATM-dependent activation of the gene encoding MAP kinase phosphatase 5 by radiomimetic DNA damage (2002) (32)
The COP9 signalosome is vital for timely repair of DNA double-strand breaks (2015) (32)
ATM AND RELATED PROTEIN KINASES : SAFEGUARDING GENOME (2003) (31)
KAP1 depletion increases PML nuclear body number in concert with ultrastructural changes in chromatin (2011) (31)
Impaired genomic stability and increased oxidative stress exacerbate different features of Ataxia-telangiectasia. (2005) (30)
In silico identification of transcriptional regulators associated with c-Myc. (2004) (29)
Molecular and cytologic analysis of DNA amplification in retinoblastoma. (1985) (29)
Condensin I recruitment and uneven chromatin condensation precede mitotic cell death in response to DNA damage (2006) (29)
Ataxia telangiectasia and lymphoma: an indication for individualized chemotherapy dosing--report of treatment in a highly inbred Arab family. (1995) (27)
Localization of an ataxia-telangiectasia locus to a 3-cM interval on chromosome 11q23: linkage analysis of 111 families by an international consortium. (1991) (26)
Investigation of the Functional Link between ATM and NBS1 in the DNA Damage Response in the Mouse Cerebellum* (2011) (25)
Assignment of the human γ-crystallin gene cluster (CRYG) to the long arm of chromosome 2, region q33–36 (1986) (25)
USP10: Friend and Foe (2010) (24)
A role for vascular deficiency in retinal pathology in a mouse model of ataxia-telangiectasia. (2011) (24)
A physical map across chromosome 11q22-q23 containing the major locus for ataxia telangiectasia. (1994) (23)
Ataxia-telangiectasia: a variant with altered in vitro phenotype of fibroblast cells. (1989) (23)
Ubiquitination capabilities in response to neocarzinostatin and H2O2 stress in cell lines from patients with ataxia-telangiectasia (2002) (22)
Conditional inactivation of the NBS1 gene in the mouse central nervous system leads to neurodegeneration and disorganization of the visual system (2009) (22)
Ataxia-telangiectasia: linkage evidence for genetic heterogeneity. (1992) (22)
The histone H 2 B-specific ubiquitin ligase RNF 20 / hBRE 1 acts as a putative tumor suppressor through selective regulation of gene expression (2008) (22)
In vitro phenotype of ataxia-telangiectasia (AT) fibroblast strains: clues to the nature of the "AT DNA lesion" and the molecular defect in AT. (1985) (21)
The cerebellar degeneration in ataxia-telangiectasia: A case for genome instability. (2020) (21)
Cells from patients with ataxia telangiectasia are abnormally sensitive to the cytotoxic effect of a tumor promoter, phorbol-12-myristate-13-acetate. (1985) (21)
Genetic toxicology of lysergic acid diethylamide (LSD-25). (1977) (20)
Paired STSs amplified from radiation hybrids, and from associated YACs, identify highly polymorphic loci flanking the ataxia telangiectasia locus on chromosome 11q22-23. (1993) (19)
The ATM protein: The importance of being active (2012) (18)
Rapid cloning of multiple amplified nucleotide sequences from human neuroblastoma cell lines by phenol emulsion competitive DNA reassociation. (1987) (18)
Rapamycin resistance in ataxia-telangiectasia. (1996) (18)
Phosphoproteomics reveals novel modes of function and inter‐relationships among PIKKs in response to genotoxic stress (2020) (18)
ATM: sounding the double-strand break alarm. (2000) (18)
Human cDNA clones that modify radiomimetic sensitivity of ataxia-telangiectasia (group A) cells (1995) (17)
The EXPANDER Integrated Platform for Transcriptome Analysis. (2019) (17)
A high-density microsatellite map of the ataxia-telangiectasia locus (1995) (17)
Three dinucleotide repeat polymorphisms at the ataxia-telangiectasia locus. (1994) (16)
Genetic Haplotyping of Ataxia-telangiectasia Families Localizes the Major Gene to an ∼ 850 kb Region on Chromosome 11q23.1. (1994) (16)
Genome instability: Linking ageing and brain degeneration (2017) (16)
Astrocyte Dysfunction Associated with Cerebellar Attrition in a Nijmegen Breakage Syndrome Animal Model (2011) (16)
A haplotype common to intermediate radiosensitivity variants of ataxia-telangiectasia in the UK. (1994) (15)
G2 chromosomal radiosensitivity in families with ataxia-telangiectasia (1989) (15)
Novel DNA sequences at chromosome 10q26 are amplified in human gastric carcinoma cell lines: molecular cloning by competitive DNA reassociation (1991) (15)
Physical localization of microsatellite markers at the ataxia-telangiectasia locus at 11q22-q23. (1994) (15)
In search of drug treatment for genetic defects in the DNA damage response: the example of ataxia-telangiectasia. (2004) (15)
Origins of Hyperphenylalaninemia in Israel (1994) (14)
Defective potassium currents in ataxia telangiectasia fibroblasts. (1998) (14)
A missense mutation, S349P, completely inactivates phenylalanine hydroxylase in North African Jews with phenylketonuria (1993) (14)
Absence of mutations in ATM, the gene responsible for ataxia telangiectasia in patients with cerebellar ataxia (1999) (14)
Repair of potentially lethal and sublethal damage induced by neocarzinostatin in normal and ataxia-telangiectasia skin fibroblasts. (1983) (14)
Maintaining integrity (2004) (13)
Malfunctioning DNA Damage Response (DDR) Leads to the Degeneration of Nigro-Striatal Pathway in Mouse Brain (2012) (13)
FBXO31: A New Player in the Ever-Expanding DNA Damage Response Orchestra (2009) (13)
Simultaneous identification and quantification of proteins by differential (16)O/(18)O labeling and UPLC-MS/MS applied to mouse cerebellar phosphoproteome following irradiation. (2009) (12)
The defect in the AT-like hamster cell mutants is complemented by mouse chromosome 9 but not by any of the human chromosomes. (1996) (12)
Inactive Atm abrogates DSB repair in mouse cerebellum more than does Atm loss, without causing a neurological phenotype. (2018) (12)
Nuclear poly(A)-binding protein 1 is an ATM target and essential for DNA double-strand break repair (2017) (12)
The Role of E3, E4 Ubiquitin Ligase (UBE4B) in Human Pathologies (2019) (12)
Phenylketonuria: variable phenotypic outcomes of the R261Q mutation and maternal PKU in the offspring of a healthy homozygote. (1993) (12)
Inhibition of TGFbeta1 Signaling Attenutates ATM Activity inResponse to Genotoxic Stress (2006) (12)
Similar repair of O6-methylguanine in normal and ataxia-telangiectasia fibroblast strains. Deficient repair capacity of lymphoblastoid cell lines does not reflect a genetic polymorphism. (1983) (12)
Ataxia-telangiectasia: Linkage analysis in highly inbred Arab and Druze families and differentiation from an ataxia-microcephaly-cataract syndrome (1992) (11)
Studying the cerebellar DNA damage response in the tissue culture dish (2013) (11)
Importance of phenotypic and molecular characterization for identification of a neuroepithelioma tumor cell line, NUB-20. (1990) (11)
Reduced inhibition of replicon initiation and chain elongation by neocarzinostatin in skin fibroblasts from patients with ataxia telangiectasia. (1982) (11)
Assignment of the human y-crystallin gene cluster ( CRYG ) to the long arm of chromosome 2 , region q 33-36 (11)
Genetic haplotyping of ataxia-telangiectasia families localizes the major gene to an approximately 850 kb region on chromosome 11q23.1. (1994) (10)
A YAC contig spanning the ataxia-telangiectasia locus (groups A and C) at 11q22-q23. (1994) (10)
A defective splice site at the phenylalanine hydroxylase gene in phenylketonuria and benign hyperphenylalaninemia among Palestinian Arabs (1992) (10)
Regulation of MRE11A by UBQLN4 leads to cisplatin resistance in patients with esophageal squamous cell carcinoma (2021) (9)
PIVOT : Protein Interactions VisualizatiOn Tool (2003) (9)
Duchenne muscular dystrophy and idiopathic hyperCKemia segregating in a family. (1995) (9)
Cytogenetic investigation of leukemic and preleukemic disorders. (1979) (9)
Analysis of the relationships between ATM and the Rad54 paralogs involved in homologous recombination repair. (2009) (9)
Ataxia-Telangiectasia: Studies on DNA Repair Synthesis in Fibroblast Strains (1981) (8)
Inactivation of phenylalanine hydroxylase by a missense mutation, R270S, in a Palestinian kinship with phenylketonuria. (1993) (8)
Novel exonic mutation (5319 G to A) resulting in two aberrantly spliced transcripts of the ATM gene in a Japanese patient with ataxia‐telangiectasia (1998) (8)
Medical genetics in Israel. (1989) (8)
Two RFLPs identified by a human chromosome #2 clone at 2p15-2p16 (HGM8 assignment no. D2S5). (1985) (8)
Atm Knockin Mice Harboring an In-frame Deletion Corresponding to the Human ATM 7636 del 9 Common Mutation Exhibit a Variant Phenotype 1 (2001) (8)
Ataxia-telangiectasia, ATM and genomic stability: maintaining a delicate balance. Two international workshops on ataxia-telangiectasia, related disorders and the ATM protein. (1998) (7)
A Haplotype Common to Intermediate Radiosensitivity Variants of Ataxia-telangiectasia in the UK. (1994) (7)
A 22‐bp deletion in the phenylalanine hydroxylase gene causing phenylketonuria in an Arab family (1992) (6)
Novel DNA rearrangement phenomena associated with DNA amplification in human neuroblastomas and neuroblastoma cell lines. (1986) (5)
Physical and genetic mapping at the ATA/ATC locus on chromosome 11q22-23. (1994) (5)
DNA damage response , bioenergetics , and neurological disease : The challenge of maintaining brain health in an aging human population (2013) (5)
An improved technique of preparing bone-marrow specimens for cytogenetic analysis (1978) (4)
DNA damage response, bioenergetics, and neurological disease: The challenge of maintaining brain health in an aging human☆,☆☆,★ (2013) (4)
Complementation of the Cellular A-T Phenotype by Gene Transfer. (1993) (4)
Use of dystrophin genomic and cDNA probes for solving difficulties in carrier detection and prenatal diagnosis of Duchenne muscular dystrophy. (1992) (3)
ATM and lymphoid malignancies; use of oriented peptide libraries to identify novel substrates of ATM critical in downstream signaling pathways. (1999) (3)
Degradation Targeting MdmX for Mdm 2-Dependent MdmX at Serine 367 Activates p 53 by DNA Damage-Induced Phosphorylation of (2005) (3)
An apparent correlation between the inhibition of induced ornithine decarboxylase activity by γ radiation and the capacity for DNA repair synthesis in normal and ataxia telangiectasia human fibroblasts: No correlation with cell survival (1981) (3)
Faculty Opinions recommendation of Functional transcription promoters at DNA double-strand breaks mediate RNA-driven phase separation of damage-response factors. (2019) (3)
The COP 9 signalosome is vital for timely repair of DNA double-strand breaks (2015) (3)
DNA sequences amplified in cancer cells: an interface between tumor biology and human genome analysis. (1992) (2)
Genetic haplotyping of ataxia-telangiectasia families localizes the major gene to an ~850 kb region on chromosome 11 q23.1. (1994) (2)
Chromosome Breakage and Sensitivity to DNA Breaking Agents in Ataxia-Telangiectasia and their Possible Association with Predisposition to Cancer (1983) (2)
The human T cell antigen Leu-2 (T8) is encoded on chromosome 2 (2004) (2)
ATM: genome stability, neuronal development and cancer cross paths (2001) (2)
Region at 11 q 23 . 3 of Heterozygosity in Breast Cancer : Identification of a New Definition and Refinement of Chromosome 11 Regions of Loss Updated (2006) (1)
PL 10 Ataxia-telangiectasia and the ATM protein: DNA damage and more (1997) (1)
3rd US-EU workshop: systems level understanding of DNA damage responses. (2010) (1)
Monitoring the ATM-Mediated DNA Damage Response in the Cerebellum Using Organotypic Cultures. (2017) (1)
DNA-based detection of chromosome deletion and amplification: diagnostic and mechanistic significance. (1986) (1)
A molecular survey of Israeli Duchenne and Becker muscular dystrophy patients. (1994) (1)
The hallmarks of aging in Ataxia-Telangiectasia (2022) (1)
Abstracts of the 15th Annual Meeting of the Israel Society for Neuroscience Eilat, Israel, December 3–5, 2006 (2006) (1)
It takes three to the DNA damage response tango (2021) (1)
, expression putative tumor suppressor through selective regulation of gene The histone H 2 B-specific ubiquitin ligase RNF 20 / hBRE 1 acts as a data (2008) (1)
RELATIVELY LOW PROPORATION OF DYSTROPHIN GENE DELETIONS IN ISRAEL DUCHENNE AND BECKER MUSCULAR DYSTROPHY PATIENTS (1994) (1)
Cloning and expression of large mammalian cDNAs: lessons from ATM. (1998) (1)
Genetic, physical and functional analysis of the ataxia-telangiectasia locus on chromosome 11q22-23 (1994) (1)
Molecular Oncology: The ATM-mediated DNA-damage response (2013) (1)
Increased Oxidative Stress in Ataxia Telangiectasia Evidenced by Alterations in Redox State of Brains from Atm-deficient Mice 1 (2001) (1)
UBQLN4 promotes STING proteasomal degradation during cisplatin‐induced DNA damage in triple‐negative breast cancer (2022) (1)
Physical and Genetic Mapping at the ATA/ATC Locus on Chromosome 11q22-23. (1994) (1)
Localization ofan Ataxia-Telangiectasia Locustoa 3-cMInterval on ChromosomeIIq23:Linkage Analysis ofIIIFamilies byan International Consortium (1991) (0)
Faculty Opinions recommendation of Mice with hyper-long telomeres show less metabolic aging and longer lifespans. (2020) (0)
Faculty Opinions recommendation of ATM activity in T cells is critical for immune surveillance of lymphoma in vivo. (2020) (0)
Abstract LB218: MRE11A expression regulated by UBQLN4 is related to cisplatin-resistance and survival in patients with esophageal squamous cell carcinoma (2021) (0)
Special issue title (2013) (0)
Genomic Amplification of UBQLN4 Is a Prognostic and Treatment Resistance Factor (2022) (0)
344 The ATM-mediated DNA damage response: back to DNA repair (2010) (0)
Physical and genetic mapping at the ATA/ATC locus on chromosome 11q22-23. (1994) (0)
Identification of a Neuroepithelioma Tumor Cell Line , NUB-20 Importance of Phenotypic and Molecular Characterization for Updated Version (2006) (0)
Faculty Opinions recommendation of Repeat expansions confer WRN dependence in microsatellite-unstable cancers. (2020) (0)
Fifth International Workshop on Ataxia-Telangiectasia 1 (1993) (0)
The serendipitous dawn of DNA repair (2020) (0)
Chapter 8 Ataxia-Telangiectasia (2007) (0)
Faculty Opinions recommendation of DNA damage kinase signaling: checkpoint and repair at 30 years. (2019) (0)
Cancer genetics: Tumor biology and genome technology converge (1993) (0)
ATM: conveying signals from damaged molecules (2000) (0)
The EXPANDER for Transcriptom (2019) (0)
9: The ATM-mediated DNA damage response: Implications for cancer development and treatment (2014) (0)
318 The ATM-mediated DNA damage response: the system and the pathways (2010) (0)
[Amplification of protooncogenes and expression of the HER2/neu oncogene in invasive breast cancer]. (1996) (0)
The role of phosphorylation in mediating cellular responses to DNA damage: the ATM-mediated DNA damage response (2011) (0)
Abstract P4-01-11: UBQLN4 regulates cisplatin-resistance in triple-negative breast cancer by targeting BAT3 for proteasomal degradation (2022) (0)
Genetic Mapping of X-Iinked Albinism-Deafness Syndrome ( ADFN ) to Xq 26 . 3q 27 . 1 (2006) (0)
Malfunctioning DNA Damage Response (DDR) Leads to the Degeneration of Nigro-Striatal Pathway in Mouse Brain (2011) (0)
Ataxia-telangiectasia and the ATM gene New insights into cellular responses to damaging agents (1996) (0)
UBQLN 4 Represses Homologous Recombination and Is Overexpressed in Aggressive Tumors Graphical (2018) (0)

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